Professor, Neurology, VA Comm Univ, Med Coll of VA Sch of Med
Neurology
Dr. William F. Maragos, MD
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William F. Maragos practices Neurology. He researched medicine at Northwestern University Feinberg School of Medicine. Northwestern University Feinberg School of Medicine has a rank of 19 in research and a 29 in primary care William Maragos is a published doctor as well. He has 49 publications published. The latest was: 6-Hydroxydopamine injections into the nigrostriatal pathway attenuate striatal malonate and 3-nitropropionic acid lesions.
Publications
- Protective effects of NIM811 in transient focal Cerebral ischemia suggest involvement of the mitochondrial permeability transition.
- Post-Injury Administration of Mitochondrial Uncouplers Increases Tissue Sparing and Improves Behavioral Outcome following Traumatic Brain Injury in Rodents.
- Progress in understanding basal ganglia dysfunction as a common target for methamphetamine abuse and HIV-1 neurodegeneration.
- HIV-1 neuropathogenesis: glial mechanisms revealed through substance abuse.
- NeuroAIDS, drug abuse, and inflammation: building collaborative research activities.
- Inhibition of tumor necrosis factor-alpha signaling prevents human immunodeficiency virus-1 protein Tat and methamphetamine interaction.
- Human immunodeficiency virus-1 protein tat and methamphetamine interactions.
- Involvement of cytokines in human immunodeficiency virus-1 protein Tat and methamphetamine interactions in the striatum.
- Methamphetamine and human immunodeficiency virus protein Tat synergize to destroy dopaminergic terminals in the rat striatum.
- Proteomic analysis of protein expression and oxidative modification in r6/2 transgenic mice: a model of Huntington Disease.
- The uncoupling agent 2,4-dinitrophenol improves mitochondrial homeostasis following striatal quinolinic acid injections.
- The mitochondrial uncoupler 2,4-dinitrophenol attenuates tissue damage and improves mitochondrial homeostasis following transient focal Cerebral ischemia.
- Enhanced Toxicity to the catecholamine tyramine in polyglutamine transfected SH-SY5Y cells.
- The mitochondrial uncoupling agent 2,4-dinitrophenol improves mitochondrial function, attenuates oxidative damage, and increases white matter sparing in the contused s...
- Mitochondrial uncoupling as a potential therapeutic target in acute central nervous system injury.
- Striatal damage and oxidative stress induced by the mitochondrial toxin malonate are reduced in clorgyline-treated rats and MAO-A deficient mice.
- HIV-1 protein Tat potentiation of methamphetamine-induced decreases in evoked overflow of dopamine in the striatum of the rat.
- Pre- or post-treatment with the mitochondrial uncoupler 2,4-dinitrophenol attenuates striatal quinolinate lesions.
- Neuronal injury in hippocampus with human immunodeficiency virus transactivating protein, Tat.
- Methamphetamine potentiates HIV-1 Tat protein-mediated activation of redox-sensitive pathways in Discrete regions of the brain.
- Human immunodeficiency virus-1 Tat protein and methamphetamine interact synergistically to impair striatal dopaminergic function.
- Molecular basis for interactions of HIV and drugs of abuse.
- Methamphetamine-induced TNF-alpha gene expression and activation of AP-1 in Discrete regions of mouse brain: potential role of reactive oxygen intermediates and lipid ...
- Mitochondrial toxin inhibition of (3)Hdopamine uptake into rat striatal synaptosomes.
- The neurologic complications of scleromyxedema.
- Estrogen protects against the synergistic Toxicity by HIV proteins, methamphetamine and cocaine.
- Acceleration of HIV Dementia with methamphetamine and cocaine.
- Neurotoxicity and dysfunction of dopaminergic systems associated with AIDS Dementia.
- Dopamine induces proteasome inhibition in neural PC12 cell line.
- NeuroToxicity of HIV-1 proteins gp120 and Tat in the rat striatum.
- Methamphetamine Toxicity is attenuated in mice that overexpress human manganese superoxide dismutase.
- Neuronal cell death in Huntington''s Disease: a potential role for dopamine.
- Clorgyline and deprenyl attenuate striatal malonate and 3-nitropropionic acid lesions.
- Evidence that Par-4 participates in the pathogenesis of HIV encephalitis.
- Does dopamine contribute to striatal damage caused by impaired mitochondrial function?
- 6-Hydroxydopamine injections into the nigrostriatal pathway attenuate striatal malonate and 3-nitropropionic acid lesions.
- The mitochondrial inhibitor malonate enhances NMDA Toxicity in the neonatal rat striatum.
- Inhibition of nitric oxide synthase activity attenuates striatal malonate lesions in rats.
- Resistance to nitroprusside neuroToxicity in perinatal rat brain.
- Neurotransmitter receptors in Alzheimer Disease.
- A study of cortical and hippocampal NMDA and PCP receptors following selective cortical and subcortical lesions.
- Excitatory amino acid binding sites in the hippocampal region of Alzheimer''s Disease and other dementias.
- Quantitative autoradiographic localization of NMDA, quisqualate and PCP receptors in the frog tectum.
- Neurons of origin and fiber trajectory of amygdalofugal projections to the medial preoptic area in Syrian hamsters.
- Anatomic correlation of NMDA and 3H-TCP-labeled receptors in rat brain.
- Glutamate transmission and toxicity in Alzheimer''s Disease.
- Loss of hippocampal 3HTCP binding in Alzheimer''s Disease.
- High correlation between the localization of 3HTCP binding and NMDA receptors.
Schools
Northwestern University The Feinberg School Of Medicine
University Of Mi Hosps
Procedures Preformed
- EMG (Electromyography)
Conditions Treated
- Alzheimer's Disease
- Cerebral Artery Thrombosis
- Cerebrovascular Disease
- Convulsions
- View All
Doctors Specialties
- Neurology
Accepted Insurances
Education
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University of Michigan Hospitals and Health Centers -
St Joseph Mercy Livingston Hospital -
Northwestern University Feinberg School of Medicine
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