Assistant Professor of Medicine, Johns Hopkins University School of Medicine
Internal Medicine
Dr. Venkataramana K. Sidhaye, MD
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Venkataramana Sidhaye practices Internal Medicine. She graduated from Northwestern University Feinberg School of Medicine. Northwestern University Feinberg School of Medicine is ranked 19/29 in Research/PrimaryCare. She has 1 awards "CMS Meaningful Use Stage 1 Certification". Doctor Venkataramana K. Sidhaye, MD is also a published MD. She has 18 paper published. The most recent paper is: Response to Letter to the Editor: Modulation of AQP-3 in burn wound: Comment on "Epidermal aquaporin-3 is increased in the cutaneous burn wound". She accepts Medicare payments and is registered with Medicare.gov.
Publications
- Response to Letter to the Editor: Modulation of AQP-3 in burn wound: Comment on "Epidermal aquaporin-3 is increased in the cutaneous burn wound".
- Epidermal aquaporin-3 is increased in the cutaneous burn wound.
- Regulatory T Cell DNA Methyltransferase Inhibition Accelerates Resolution of Lung Inflammation.
- Foxp3(+) regulatory T cells promote Lung epithelial proliferation.
- Mitogen-Activated Protein Kinase-Activated Protein Kinase 2 Mediates Apoptosis during Lung Vascular Permeability by Regulating Movement of Cleaved Caspase 3.
- Immunological Priming Requires Regulatory T Cells and IL-10-Producing Macrophages To Accelerate Resolution from Severe Lung Inflammation.
- Aquaporin 5 regulates cigarette smoke induced emphysema by modulating barrier and immune properties of the epithelium.
- Effect of modifying quantum dot surface charge on airway epithelial cell uptake in vitro.
- Macrophage A2A Adenosinergic Receptor (ADORA2A) Modulates Oxygen-Induced Augmentation of Murine Lung Injury.
- Regulatory T cells reduce acute Lung injury fibroproliferation by decreasing fibrocyte recruitment.
- Aquaporin 5 polymorphisms and rate of lung function decline in Chronic obstructive pulmonary disease.
- cAMP has distinct acute and Chronic effects on aquaporin-5 in lung epithelial cells.
- Accumulation of aquaporin-1 during hemolysin-induced necrotic cell death.
- Transient receptor potential vanilloid 4 regulates aquaporin-5 abundance under hypotonic conditions.
- Septin-2 mediates airway epithelial barrier function in physiologic and pathologic conditions.
- Moderate oxygen augments lipopolysaccharide-induced Lung injury in mice.
- Shear stress regulates aquaporin-5 and airway epithelial barrier function.
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Atelectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
- Internal Medicine
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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Johns Hopkins University -
McGaw Medical Center of Northwestern University -
Northwestern University Feinberg School of Medicine
Hospital
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Johns Hopkins Hospital -
Johns Hopkins Bayview Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1205805751 |
| Last Name Of The Provider | SIDHAYE |
| First Name Of The Provider | VENKATARAMANA |
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