Professor of Medicine, University Alabama
Pulmonology
Dr. Veena B. Antony, MD
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Veena Antony works in the field of Pulmonology. She went to medical school at Christian Medical College Ludhiana. She has 1 awards "America's Top Doctors". Dr. Veena B. Antony is also a published doctor. She has 92 publication published. The most recent publication is: Interleukin 8: an autocrine growth factor for malignant mesothelioma. Doctor Veena B. Antony, MD is registered with Medicare and accepts Medicare payments.
Publications
- Blue journal conference. Aging and susceptibility to Lung disease.
- Noninvasive Imaging of Experimental Lung Fibrosis.
- Plasminogen activator inhibitor 1, fibroblast apoptosis resistance, and aging-related susceptibility to Lung fibrosis.
- Wilms' tumor 1 (Wt1) regulates pleural mesothelial cell plasticity and transition into myofibroblasts in idiopathic Pulmonary fibrosis.
- The effect of biomass fuel exposure on the prevalence of Asthma in adults in India - review of current evidence.
- miR-145 regulates myofibroblast differentiation and Lung fibrosis.
- Pleural Mesothelial Cell Differentiation and Invasion in Fibrogenic Lung Injury.
- Silencing receptor EphA2 induces apoptosis and attenuates tumor growth in malignant mesothelioma.
- Targeted delivery of amikacin into granuloma.
- Pleural mesothelial cell transformation into myofibroblasts and haptotactic migration in response to TGF-beta1 in vitro.
- Innate immune responses in murine Pleural mesothelial cells: Toll-like receptor-2 dependent induction of beta-defensin-2 by staphylococcal peptidoglycan.
- Bacterial induction of early response genes and activation of proapoptotic factors in Pleural mesothelial cells.
- Induction of cell cycle arrest and apoptosis by BCG Infection in cultured human bronchial airway epithelial cells.
- Cyclic stretch induces PlGF expression in bronchial airway epithelial cells via nitric oxide release.
- Silencing the receptor EphA2 suppresses the growth and haptotaxis of malignant mesothelioma cells.
- Pleural Fibrosis.
- The mysterious Pulmonary brush cell: a cell in search of a function.
- Talc pleurodesis: basic fibroblast growth factor mediates pleural Fibrosis.
- RSV causes HIF-1alpha stabilization via NO release in primary bronchial epithelial cells.
- Th2 cytokines IL-4 and IL-13 downregulate paxillin expression in bronchial airway epithelial cells.
- Respiratory syncytial virus causes increased bronchial epithelial permeability.
- Defensive role of Pleural mesothelial cell sialomucins in tumor metastasis.
- Pleural mesothelial cells modulate polymorphonuclear leukocyte apoptosis in empyema.
- Pleural mesothelial cell (PMC) defense mechanisms against malignancy.
- Fibrinolysis in the Pleural space: breaking the bonds that bind.
- Induction of MCP-1 expression in airway epithelial cells: role of CCR2 receptor in airway epithelial injury.
- Inflammatory cytokines mediate C-C (monocyte chemotactic protein 1) and C-X-C (interleukin 8) chemokine expression in human Pleural fibroblasts.
- Low molecular weight hyaluronan induces malignant mesothelioma cell (MMC) proliferation and haptotaxis: role of CD44 receptor in MMC proliferation and haptotaxis.
- Adherence of ovarian Cancer cells induces pleural mesothelial cell (PMC) permeability.
- SMAD-independent down-regulation of caveolin-1 by TGF-β: effects on proliferation and survival of myofibroblasts.
- [Autonomic changes during transitional phases of Sleep in the normal adult].
- Daidzin, an antioxidant isoflavonoid, decreases blood alcohol levels and shortens Sleep time induced by ethanol intoxication.
- Management of malignant Pleural effusions.
- Bacterial induction of Pleural mesothelial monolayer barrier dysfunction.
- Polar production of interleukin-8 by mesothelial cells promotes the transmesothelial migration of neutrophils: role of intercellular adhesion molecule-1.
- Induction of acute Pleural inflammation by Staphylococcus aureus. I. CD4+ T cells play a critical role in experimental empyema.
- MCP-1 in Pleural injury: CCR2 mediates haptotaxis of Pleural mesothelial cells.
- Talc induces apoptosis in human malignant mesothelioma cells in vitro.
- Pathogenesis of malignant Pleural effusions and talc pleurodesis.
- Inhibition of interleukin-8 reduces human malignant Pleural mesothelioma propagation in nude mouse model.
- Mycobacterium-induced transmesothelial migration of monocytes into Pleural space: role of intercellular adhesion molecule-1 in tuberculous pleurisy.
- Helper T cell type 1 and 2 cytokines regulate C-C chemokine expression in mouse Pleural mesothelial cells.
- Pathophysiology of pleural space Infections.
- Interleukin 8: an autocrine growth factor for malignant mesothelioma.
- Mycobacterium-mediated chemokine expression in Pleural mesothelial cells: role of C-C chemokines in tuberculous pleurisy.
- Macrophage inflammatory protein-1alpha C-C chemokine in parapneumonic Pleural effusions.
- Talc-induced expression of C-C and C-X-C chemokines and intercellular adhesion molecule-1 in mesothelial cells.
- Drug-induced pleural Disease.
- Immunobiology of Pleural inflammation: potential implications for pathogenesis, diagnosis and therapy.
- Pleurodesis: state of the art.
- Adenosine deaminase isoenzymes and Pleural tuberculosis.
- Angiogenesis in mesotheliomas. Role of mesothelial cell derived IL-8.
- Alcohol-induced inhibition of alveolar macrophage oxidant release in vivo and in vitro.
- The immunopathology of chemotactic cytokines: the role of interleukin-8 and monocyte chemoattractant protein-1.
- Recruitment of inflammatory cells to the Pleural space. Chemotactic cytokines, IL-8, and monocyte chemotactic peptide-1 in human Pleural fluids.
- Mesothelial cell modulation of Pleural repair: thrombin stimulated mesothelial cells release prostaglandin E2.
- Pleural mesothelial cell expression of C-C (monocyte chemotactic peptide) and C-X-C (interleukin 8) chemokines.
- Testing the waters. Differentiating transudates from exudates.
- Empyema after internal jugular catheterisation in rheumatoid arthritis.
- The pathogenesis of the low Pleural fluid pH in esophageal rupture.
- Dimethyl sulfoxide inhibits phagocyte influx into infected Pleural spaces and phagocyte locomotion in vitro.
- The contribution of leukocytes and bacteria to the low pH of empyema fluid.
- The pleuroPulmonary manifestations of the postcardiac injury syndrome.
- Lupus pleuritis. Clinical features and Pleural fluid characteristics with special reference to Pleural fluid antinuclear antibodies.
- Effect of dimethyl sulfoxide on chemotaxis of phagocytic cells.
- Pathogenesis of Pleural plaques. Relationship of early cellular response and pathology.
- Inflammatory responses in experimental tuberculosis pleurisy.
- Lung repair and granuloma formation. Tubercle bacilli stimulated neutrophils release chemotactic factors for monocytes.
- Bacillus Calmette-Guérin-stimulated neutrophils release chemotaxins for monocytes in rabbit Pleural spaces and in vitro.
- Pleurodesis--testing the waters.
- Polymorphonuclear leukocyte cytoplasts mediate acute Lung injury.
- Stimulation of cAMP accumulation and superoxide production in human neutrophils and monocytes.
- Pleural mesothelial cells stimulated by asbestos release chemotactic activity for neutrophils in vitro.
- Alveolar cell population in HIV infected patients.
- NHLBI workshop summaries. Pleural cell biology in health and Disease.
- Mesothelial cell response to Pleural injury: thrombin-induced proliferation and chemotaxis of rat Pleural mesothelial cells.
- Mechanism of tetracycline-hydrochloride-induced pleurodesis. Tetracycline-hydrochloride-stimulated mesothelial cells produce a growth-factor-like activity for fibrobla...
- Immunological mechanisms in pleural Disease.
- Drawing the line: differentiating transudates from exudates.
- Mycobacteria induces Pleural mesothelial permeability by down-regulating beta-catenin expression.
- Respiratory system responsiveness in rabbits in vivo is reduced by prolonged continuous positive airway pressure.
- Primum non nocere and malignant Pleural effusions.
- Pathogenesis of pleural Infection.
- Involvement of calpain-calpastatin in cigarette smoke-induced inhibition of Lung endothelial nitric oxide synthase.
- Talc mediates angiostasis in malignant Pleural effusions via endostatin induction.
- Pathophysiology of the pleura.
- Receptor EphA2 activation with ephrinA1 suppresses growth of malignant mesothelioma (MM).
- Parenchymal trafficking of pleural mesothelial cells in idiopathic Pulmonary fibrosis.
- New insights into the pathogenesis and treatment of idiopathic Pulmonary fibrosis.
- Erythromycin improves gastric emptying half-time in adult Cystic fibrosis patients with gastroparesis.
- Thioredoxin as a biomarker for graft rejection in Lung transplant recipients.
Procedures Preformed
- Bronchoscopy
- Continuous Positive Airway Pressure (CPAP)
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Atelectasis
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Doctors Specialties
- Pulmonology
Accepted Insurances
Awards
- America's Top Doctors
Education
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University of Colorado -
Christian Medical College Ludhiana
Hospital
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University of Alabama Hospital
Drug Facts
| NPI NUMBER | 1033150057 | |
| NPPES Provider LastName | ANTONY | |
| NPPES Provider FirstName | VEENA | |
| NPPES Provider ZIPCode | 352332110 | |
| NPPES Provider State | AL | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 253.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1033150057 |
| Last Name Of The Provider | ANTONY |
| First Name Of The Provider | VEENA |
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