University of Wisconsin Hospital and Clinics
Cardiology
Dr. Timothy J. Kamp, MD
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Timothy Kamp works in the field of Cardiology. He went to University of Chicago Pritzker School of Medicine. University of Chicago Pritzker School of Medicine is ranked 10/19 in Research/PrimaryCare. He received awards:"Meaningful Use Stage 1 Certification" and "Fellow (FACC)". Dr. Timothy J. Kamp is a published doctor and has 89 publications published. The lastest was: 'Recurrent ischemic strokes in a patient with Medtronic-Hall prosthetic aortic valve and valve strands.' He accepts Medicare.
Publications
- Lineage Reprogramming of Fibroblasts into Proliferative Induced Cardiac Progenitor Cells by Defined Factors.
- Functional Effects of a Tissue-Engineered Cardiac Patch From Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes in a Rat Infarct Model.
- Harry A. Fozzard, MD: 1931–2014.
- Cardiac repair in a porcine model of Acute myocardial infarction with human induced pluripotent stem cell-derived cardiovascular cells.
- Pompe Disease results in a Golgi-based glycosylation deficit in human induced pluripotent stem cell-derived cardiomyocytes.
- Signals from the surface modulate differentiation of human pluripotent stem cells through glycosaminoglycans and integrins.
- Calcium Transients Closely Reflect Prolonged Action Potentials in iPSC Models of Inherited Cardiac Arrhythmia.
- Cardiomyopathy, mitochondria and Barth Syndrome: iPSCs reveal a connection.
- Induced pluripotent stem cells for post-Myocardial infarction repair: remarkable opportunities and challenges.
- Irx4 identifies a chamber-specific cell population that contributes to ventricular myocardium development.
- Micropattern width dependent sarcomere development in human ESC-derived cardiomyocytes.
- Directed cardiomyocyte differentiation from human pluripotent stem cells by modulating Wnt/?-catenin signaling under fully defined conditions.
- Insulin Inhibits Cardiac Mesoderm, not Mesendoderm, formation during Cardiac Differentiation of Human Pluripotent Stem Cells and Modulation of Canonical Wnt Signaling ...
- Extracellular matrix promotes highly efficient cardiac differentiation of human pluripotent stem cells: the matrix sandwich method.
- Differentiation of human embryonic stem cells and induced pluripotent stem cells to cardiomyocytes: a methods overview.
- Robust cardiomyocyte differentiation from human pluripotent stem cells via temporal modulation of canonical Wnt signaling.
- Directed Fusion of Mesenchymal Stem Cells with Cardiomyocytes via VSV-G Facilitates Stem Cell Programming.
- Nonuniform Cardiac Denervation Observed by (11)C-meta-Hydroxyephedrine PET in 6-OHDA-Treated Monkeys.
- Role of RBM25/LUC7L3 in Abnormal Cardiac Sodium Channel Splicing Regulation in Human Heart Failure.
- High Purity Human Induced Pluripotent Stem Cell (hiPSC) Derived Cardiomyocytes: Electrophysiological Properties of Action Potentials and Ionic Currents.
- Different subcellular populations of L-type Ca(2+) channels exhibit unique regulation and functional roles in cardiomyocytes.
- Imaging of Induced Pluripotent Stem Cells: From Cellular Reprogramming to Transplantation.
- Small GTPase Rab11b regulates degradation of surface membrane L-type Cav1.2 channels.
- Robust L-Type Calcium Current Expression following Heterozygous Knockout of the Cav1.2 Gene in Adult Mouse Heart.
- An electrifying iPSC disease model: long QT Syndrome type 2 and heart cells in a dish.
- Recognizing Heart cells in a crowd.
- Microfluidic cell culture and its application in high-throughput drug screening: cardiotoxicity assay for hERG channels.
- Multiphoton Flow Cytometry to Assess Intrinsic and Extrinsic Fluorescence in Cellular Aggregates: Applications to Stem Cells.
- A sympathetic model of L-type Ca2+ channel-triggered Arrhythmias.
- On the road to iPS cell cardiovascular applications.
- Functional cardiomyocytes derived from human induced pluripotent stem cells.
- Small GTPase determinants for the Golgi processing and plasmalemmal expression of human ether-a-go-go related (hERG) K+ channels.
- Conditional forebrain deletion of the L-type calcium channel Ca V 1.2 disrupts remote spatial memories in mice.
- Transplanted embryonic stem cells following mouse Myocardial infarction inhibit apoptosis and cardiac remodeling.
- Kv11.1 (ERG1) K+ channels localize in cholesterol and sphingolipid enriched membranes and are modulated by membrane cholesterol.
- Human embryonic stem cell-derived cardiomyocytes can be maintained in defined medium without serum.
- Mutant caveolin-3 induces persistent late sodium current and is associated with long-QT Syndrome.
- Mission impossible: IGF-1 and PTEN specifically "Akt"ing on cardiac L-type Ca2+ channels.
- Localization of cardiac L-type Ca(2+) channels to a caveolar macromolecular signaling complex is required for beta(2)-adrenergic regulation.
- Specific serine proteases selectively damage KCNH2 (hERG1) potassium channels and I(Kr).
- Crosstalk of beta-adrenergic receptor subtypes through Gi blunts beta-adrenergic stimulation of L-type Ca2+ channels in canine Heart failure.
- Intragenic suppression of trafficking-defective KCNH2 channels associated with long QT Syndrome.
- Unique modulation of L-type Ca2+ channels by short auxiliary beta1d subunit present in cardiac muscle.
- Increased nitration of sarcoplasmic reticulum Ca2+-ATPase in human Heart failure.
- Blocking the L-type Ca2+ channel with a gem: a paradigm for a more specific Ca2+ channel blocker.
- Images in cardiovascular medicine. Infiltrative eosinophilic myocarditis diagnosed and localized by cardiac magnetic resonance imaging.
- Molecular heterogeneity of calcium channel beta-subunits in canine and human Heart: evidence for differential subcellular localization.
- Localization of functional endothelin receptor signaling complexes in cardiac transverse tubules.
- Thapsigargin selectively rescues the trafficking Defective LQT2 channels G601S and F805C.
- Human embryonic stem cells develop into multiple types of cardiac myocytes: action potential characterization.
- L-type Ca2+ channels gaining respect in Heart failure.
- Opportunities for use of human iPS cells in predictive toxicology.
- Reduction in density of transverse tubules and L-type Ca(2+) channels in canine tachycardia-induced Heart failure.
- Regulation of cardiac L-type calcium channels by protein kinase A and protein kinase C.
- Tails of the L-type Ca(2+) channel: to sense oxygen or not.
- Hairpin properties of single-stranded DNA containing a GC-rich triplet repeat: (CTG)15.
- Transcriptional regulation of the neuronal L-type calcium channel alpha 1D subunit gene.
- Functional properties of cardiac L-type calcium channels transiently expressed in HEK293 cells. Roles of alpha 1 and beta subunits.
- Stimulation rate modulates effects of the dihydropyridine CGP 28 392 on cardiac calcium-dependent action potentials.
- Voltage-dependent nitrendipine binding to cardiac sarcolemmal vesicles.
- Voltage-dependent binding of dihydropyridine calcium channel blockers to guinea pig ventricular myocytes.
- Voltage- and use-dependent modulation of cardiac calcium channels by the dihydropyridine (+)-202-791.
- Separation of cardiac plasmalemma into cell surface and T-tubular components. Distribution of saxitoxin- and nitrendipine-binding sites.
- Calcium channel agonist and antagonist effects of the stereoisomers of the dihydropyridine 202-791.
- Myocardial infarction, Aortic dissection, and thrombolytic therapy.
- Functional consequences of sulfhydryl modification in the pore-forming subunits of cardiovascular Ca2+ and Na+ channels.
- Enhancement of ionic current and charge movement by coexpression of calcium channel beta 1A subunit with alpha 1C subunit in a human embryonic Kidney cell line.
- Endothelin-1 and photoreleased diacylglycerol increase L-type Ca2+ current by activation of protein kinase C in rat ventricular myocytes.
- Voltage-dependent facilitation of cardiac L-type Ca channels expressed in HEK-293 cells requires beta-subunit.
- High-voltage injury resulting in permanent right Heart dysfunction.
- Recurrent ischemic strokes in a patient with Medtronic-Hall prosthetic Aortic valve and valve strands.
- Defects of beta 2-adrenergic signal transduction in chronic lymphocytic leukaemia: relationship to Disease progression.
- Randomized study of the combination of hydroxyurea and interferon alpha versus hydroxyurea monotherapy during the Chronic phase of Chronic myelogenous leukemia (CML St...
- Regulated plasma levels of colony-stimulating factors, interleukin-6 and interleukin-10 in patients with Acute leukaemia and non-hodgkin''s lymphoma undergoing cytored...
- [Agranulocytosis caused by infectious-toxic bone marrow damage after Borrelia infection].
- Reduced responsiveness of adenylate cyclase to forskolin in human lymphoma cells.
- The activation of intracellular tyrosine kinases by interferon-alpha (IFNalpha) correlates with its antiproliferative activity in B-lymphoid cell lines, but not in B-c...
- L-type Ca2+ channels in Atrial fibrillation: wallflowers or a vanishing act.
- Depletion of T-tubules and specific subcellular changes in sarcolemmal proteins in tachycardia-induced Heart failure.
- Distinct mouse Coronary anatomy and myocardial infarction consequent to ligation.
- Transplantation of embryonic stem cells into the infarcted mouse Heart: formation of multiple cell types.
- Embryonic stem cells: differentiation into cardiomyocytes and potential for Heart repair and regeneration.
- Increased late sodium current in myocytes from a canine Heart failure model and from failing human Heart.
- Structure-function relationships of Ca spark activity in normal and failing cardiac myocytes as revealed by flash photography.
- Cellular therapies for heart Disease: unveiling the ethical and public policy challenges.
- Caveolae, ion channels and cardiac Arrhythmias.
- The microwell control of embryoid body size in order to regulate cardiac differentiation of human embryonic stem cells.
- Cardiomyocyte transverse tubule loss leads the way to Heart failure.
Schools
University Of ChicagoThe Pritzker School Of Medicine
Johns Hopkins Hospital
Procedures Preformed
- Ablation for Treatment of Cardiac Arrhythmias
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac Myocardial Perfusion Imaging
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Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Dissection
- Aortic Ectasia
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Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Meaningful Use Stage 1 Certification
- Fellow (FACC)
Education
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Johns Hopkins University -
University of Chicago Pritzker School of Medicine
Hospital
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University of Wisconsin Hospital and Clinics -
William S. Middleton Memorial Veterans Hospital
Drug Facts
| NPI NUMBER | 1619943602 | |
| NPPES Provider LastName | KAMP | |
| NPPES Provider FirstName | TIMOTHY | |
| NPPES Provider ZIPCode | 53792 | |
| NPPES Provider State | WI | |
| Specialty Description | Cardiology | |
| Total Claim Count | 720.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1619943602 |
| Last Name Of The Provider | KAMP |
| First Name Of The Provider | TIMOTHY |
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