Professor, Medicine, Stony Brook University SOM
Hematology
Dr. Theodore G. Gabig, MD
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Doctor Theodore G. Gabig, MD works in the field of Hematology. He researched medicine at Loyola University Chicago Stritch School of Medicine. He received 1 awards: "Elected Member". Theodore Gabig is a published physician. He published 48 publications, including: 'Expression and chromosomal localization of the Requiem gene.' Theodore Gabig accepts Medicare payments and is registered with Medicare.gov.
Publications
- Clostridium perfringens enterotoxin elicits rapid and specific cytolysis of breast carcinoma cells mediated through tight junction proteins claudin 3 and 4.
- Phagocyte NADPH-oxidase. Studies With flavin analogues as active site probes in triton X-100-solubilized preparations.
- Differentiation of cellular processes involved in the induction and maintenance of stimulated neutrophil adherence.
- The respiratory burst oxidase of human neutrophils. Further studies of the purified enzyme.
- NADPH oxidase from polymorphonuclear cells.
- Functional relationship of the cytochrome b to the superoxide-generating oxidase of human neutrophils.
- The NADPH-dependent O-.2-generating oxidase from human neutrophils.
- Proton stoichiometry Associated with human neutrophil respiratory-burst reactions.
- Catalytic properties of the resolved flavoprotein and cytochrome B components of the NADPH dependent O2- . generating oxidase from human neutrophils.
- Deficient flavoprotein component of the NADPH-dependent O2-.-generating oxidase in the neutrophils from three male patients with Chronic granulomatous disease.
- Leukocyte Abnormalities.
- The killing of pathogens by phagocytes.
- The subcellular particulate NADPH-dependent O2.(-)-generating oxidase from human blood monocytes: comparison to the neutrophil System.
- Function of wild-type or mutant Rac2 and Rap1a GTPases in differentiated HL60 cell NADPH oxidase activation.
- Requiem: a novel zinc finger gene essential for apoptosis in Myeloid cells.
- Therapeutic leukapheresis in hyperleucocytic leukaemias: lack of correlation between degree of cytoreduction and early mortality rate.
- Expression and chromosomal localization of the Requiem gene.
- Combined heterozygosity of factor V leiden and the G20210A prothrombin gene mutation in a patient with cerebral cortical Vein thrombosis.
- Prevention of arterial thromboembolism in nonvalvular atrial fibrillation: validating molecular markers of risk stratification.
- Expression of Clostridium perfringens enterotoxin receptors claudin-3 and claudin-4 in proState cancer epithelium.
- Role for c-jun N-terminal kinase in treatment-refractory Acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation.
- Deactivation of the subcellular NADPH oxidase and its relationship to termination of the respiratory burst.
- Expression of val-12 mutant ras p21 in an IL-3-dependent murine Myeloid cell line is associated with loss of serum-dependence and increases in membrane PIP2-specific p...
- Resolution of a low molecular weight G protein in neutrophil cytosol required for NADPH oxidase activation and reconstitution by recombinant Krev-1 protein.
- Growth factor-dependent initiation of DNA replication in nuclei isolated from an interleukin 3-dependent murine Myeloid cell line.
- A neutrophil GTP-binding protein that regulates cell free NADPH oxidase activation is located in the cytosolic fraction.
- Purification and characterization of a lipid thiobis ester from human neutrophil cytosol that reversibly deactivates the O2- -generating NADPH oxidase.
- A RAS oncogene imparts growth factor independence to Myeloid cells that abnormally regulate protein kinase C: a nonautocrine transformation pathway.
- Reversible activation of the neutrophil superoxide generating System by hexachlorocyclohexane: correlation with effects on a subcellular superoxide-generating fraction.
- A mutant RAS gene acts through protein kinase C to augment interleukin-3 dependent proliferation in a fastidious immortal Myeloid cell line.
- Effects of oxygen tension and pH on the respiratory burst of human neutrophils.
- Evidence for production of oxidizing radicals by the particulate O-2-forming System from human neutrophils.
- The O2(-) -forming oxidase responsible for the respiratory burst in human neutrophils. Properties of the solubilized enzyme.
- Solubilization of the O2(-)-forming activity responsible for the respiratory burst in human neutrophils.
- MOZ and MOZ-CBP cooperate With NF-kappaB to activate transcription from NF-kappaB-dependent promoters.
- Temporal gene expression profile of human precursor B Leukemia cells induced by adhesion receptor: identification of pathways regulating B-cell survival.
- Simultaneous mobilization of Mac-1 (CD11b/CD18) and formyl peptide chemoattractant receptors in human neutrophils.
- The effects of oxidizing species derived from molecular oxygen on the proliferation in vitro of human granulocyte-macrophage progenitor cells.
- Rapid deactivation of NADPH oxidase in neutrophils: continuous replacement by newly activated enzyme sustains the respiratory burst.
- Isolation of the respiratory burst oxidase: the role of a flavoprotein component.
- Activation of the human neutrophil NADPH oxidase results in coupling of electron carrier function between ubiquinone-10 and cytochrome b559.
- Molecular heterogeneity in Chronic granulomatous disease: a human model of defective phagocyte superoxide production.
- Regulation of neutrophil NADPH oxidase activation in a cell-free System by guanine nucleotides and fluoride. Evidence for participation of a pertussis and cholera toxi...
- Relationship of phosphatidylinositol bisphosphate hydrolysis to calcium mobilization and functional activation in fluoride-treated neutrophils.
- Cell-free activation of phagocyte NADPH-oxidase: tissue and differentiation-specific expression of cytosolic cofactor activity.
- Temporal adaptation of neutrophil oxidative responsiveness to n-formyl-methionyl-leucyl-phenylalanine. Acceleration by granulocyte-macrophage colony stimulating factor.
- Restoration of adenylate cyclase responsiveness in murine myeloid Leukemia permits inhibition of proliferation by hormone. Butyrate augments catalytic activity of aden...
Schools
Loyola University of Chicago Stritch School of Medicine
Procedures Preformed
- HPC Transplantation (Stem Cell Transplant)
- Plasmapheresis
Conditions Treated
- Acute Leukemia
- Anemia
- Antiphospholipid Syndrome (APS)
- Burkitt's Lymphoma
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Doctors Specialties
- Hematology
Accepted Insurances
Awards
- Elected Member
Education
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Tufts Medical Center -
University of Chicago -
Loyola University Chicago Stritch School of Medicine
Hospital
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Stony Brook University Medical Center
Drug Facts
| NPI NUMBER | 1538194360 | |
| NPPES Provider LastName | GABIG | |
| NPPES Provider FirstName | THEODORE | |
| NPPES Provider ZIPCode | 11794 | |
| NPPES Provider State | NY | |
| Specialty Description | Hematology/Oncology | |
| Total Claim Count | 107.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 12.0 | |
| Percent Opioid Claims | 11.21 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1538194360 |
| Last Name Of The Provider | GABIG |
| First Name Of The Provider | THEODORE |
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