Resident Physician, Pathology, UCSF School of Medicine
Pathology
Dr. Sarah E. Umetsu, MD
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Sarah Umetsu is an expert in the field of Pathology. Sarah E. Umetsu is also published. She has 15 publications published. The lastest publication: "TIM-4, expressed by medullary macrophages, regulates respiratory tolerance by mediating phagocytosis of antigen-specific T cells.'
Publications
- TIM-4, expressed by medullary macrophages, regulates respiratory tolerance by mediating phagocytosis of antigen-specific T cells.
- TIM-4, a receptor for phosphatidylserine, controls adaptive immunity by regulating the removal of antigen-specific T cells.
- Apoptotic cells activate NKT cells through T cell Ig-like mucin-like-1 resulting in airway hyperreactivity.
- Notch target gene deregulation and maintenance of the leukemogenic phenotype do not require RBP-J kappa in Ikaros null mice.
- Ikaros is a regulator of Il10 expression in CD4+ T cells.
- Cutting edge: Ikaros is a regulator of Th2 cell differentiation.
- TIM gene family and their role in atopic diseases.
- TIM-1 and TIM-4 glycoproteins bind phosphatidylserine and mediate uptake of apoptotic cells.
- Ikaros enforces the costimulatory requirement for IL2 gene expression and is required for anergy induction in CD4+ T lymphocytes.
- Immunoglobulin A (IgA) is a natural ligand of hepatitis A virus cellular receptor 1 (HAVCR1), and the association of IgA with HAVCR1 enhances virus-receptor interactions.
- TIM-4 is the ligand for TIM-1, and the TIM-1-TIM-4 interaction regulates T cell proliferation.
- TIM-1 induces T cell activation and inhibits the development of peripheral tolerance.
- Immunology: hepatitis A virus link to atopic disease.
- Identification of Tapr (an airway hyperreactivity regulatory locus) and the linked Tim gene family.
Schools
Northwestern Center Feinberg School of Medicine
Doctors Specialties
- Pathology
Accepted Insurances
Education
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UCSF
Hospital
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UCSF Medical Center
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