Professor, Medicine, Medical College of Wisconsin
Hematology
Dr. Roy L. Silverstein, MD
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Roy L. Silverstein practices Hematology. He attended Emory University School of Medicine. Emory University School of Medicine has a rank of 23 in research and a 42 in primary care He over the years received 1 awards: "Member". Dr. Roy L. Silverstein is also a published physician. He has 58 publication published. The most recent publication is: Extracellular Vesicles Activate a CD36-Dependent Signaling Pathway to Inhibit Microvascular Endothelial Cell Migration and Tube Formation.
Publications
- Extracellular Vesicles Activate a CD36-Dependent Signaling Pathway to Inhibit Microvascular Endothelial Cell Migration and Tube Formation.
- Atherothrombosis: Seeing Red?
- Thrombospondin-1 restrains neutrophil granule serine protease function and regulates the innate immune response during Klebsiella pneumoniae infection.
- Thymidine Phosphorylase Participates in Platelet Signaling and Promotes Thrombosis.
- Hyper-Inflammation and Skin Destruction Mediated by Rosiglitazone Activation of Macrophages in IL-6 Deficiency.
- Exosome poly-ubiquitin inhibits platelet activation, downregulates CD36, and inhibits pro-atherothombotic cellular functions.
- Platelet CD36 Links Not Only α-Granule-Derived Proteins to Thrombus Stability But Also Metabolic and Oxidant Stress to a Prothrombotic Phenotype.
- Cancer stem cell-specific scavenger receptor CD36 drives glioblastoma progression.
- Proteasome Proteolysis Supports Stimulated Platelet Function and Thrombosis.
- Thrombospondin-1 triggers macrophage IL-10 production and promotes resolution of experimental lung injury.
- Lipopolysaccharide Stimulates Platelets through an IL-1β Autocrine Loop.
- Vav Guanine nucleotide exchange factors regulate atherosclerotic lesion development in mice.
- Molecular basis of antiangiogenic thrombospondin-1 type 1 repeat domain interactions with CD36.
- Thrombospondin-1 modulates VEGF signaling via CD36 by recruiting SHP-1 to VEGFR2 complex in microvascular endothelial cells.
- CD36 and Na/K-ATPase-?1 form a proinflammatory signaling loop in kidney.
- CD36 Ectodomain Phosphorylation Blocks Thrombospondin-1 Binding: Structure-Function Relationships and Regulation by Protein Kinase C.
- Teaching an old dog new tricks: potential antiatherothrombotic use for statins.
- Vav GEF regulates CD36-mediated macrophage foam cell formation via calcium and dynamin-dependent processes.
- Expression, purification and structural characterization of functionally replete thrombospondin-1 type 1 repeats in a bacterial expression system.
- Loss of expression of neutrophil proteinase-3: a contributory factor in thrombotic risk in paroxysmal nocturnal hemoglobinuria.
- Platelet CD36 surface expression levels affect functional responses to oxidized LDL and are associated with inheritance of specific genetic polymorphisms.
- Lysophosphatidic acid suppresses endothelial cell CD36 expression and promotes angiogenesis via a PKD-1 dependent signaling pathway.
- Vav guanine nucleotide exchange factors link hyperlipidemia and a prothrombotic state.
- Vav family Rho guanine nucleotide exchange factors regulate CD36-mediated macrophage foam cell formation.
- CD9 Tetraspanin Interacts with CD36 on the Surface of Macrophages: A Possible Regulatory Influence on Uptake of Oxidized Low Density Lipoprotein.
- CD36 participates in a signaling pathway that regulates ROS formation in murine VSMCs.
- Histidine-rich glycoprotein modulates the anti-angiogenic effects of vasculostatin.
- Mechanisms of cell signaling by the scavenger receptor CD36: implications in atherosclerosis and thrombosis.
- Lipid-induced insulin resistance is associated with increased monocyte expression of scavenger receptor CD36 and internalization of oxidized LDL.
- Type 2 scavenger receptor CD36 in platelet activation: the role of hyperlipemia and oxidative stress.
- Inflammation, atherosclerosis, and arterial thrombosis: role of the scavenger receptor CD36.
- Deficiency of LRP8 in mice is associated with altered platelet function and prolonged time for in vivo thrombosis.
- Vasculostatin inhibits intracranial glioma growth and negatively regulates in vivo angiogenesis through a CD36-dependent mechanism.
- Thrombospondin-1-induced apoptosis of brain microvascular endothelial cells can be mediated by TNF-R1.
- CD36 modulates migration of mouse and human macrophages in response to oxidized LDL and may contribute to macrophage trapping in the arterial intima.
- TLR2 and its co-receptors determine responses of macrophages and dendritic cells to lipoproteins of Mycobacterium tuberculosis.
- CD36, a scavenger receptor involved in immunity, metabolism, angiogenesis, and behavior.
- Biological surgery: synergetic angiogenic therapy using coadministration of two progenitor cell populations.
- A specific CD36-dependent signaling pathway is required for platelet activation by oxidized low-density lipoprotein.
- Platelet CD36 mediates interactions with endothelial cell-derived microparticles and contributes to thrombosis in mice.
- Hepatic fatty acid transporter Cd36 is a common target of LXR, PXR, and PPARgamma in promoting steatosis.
- Venous thrombosis in the elderly: more questions than answers.
- Platelet CD36 links hyperlipidemia, oxidant stress and a prothrombotic phenotype.
- Neprilysin inhibits angiogenesis via proteolysis of fibroblast growth factor-2.
- A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.
- Light-induced oxidation of photoreceptor outer segment phospholipids generates ligands for CD36-mediated phagocytosis by retinal pigment epithelium: a potential mechan...
- EP 80317, a ligand of the CD36 scavenger receptor, protects apolipoprotein E-deficient mice from developing atherosclerotic lesions.
- The class B scavenger receptor CD36 mediates free radical production and tissue injury in cerebral ischemia.
- Stem cell transplantation reveals that absence of macrophage CD36 is protective against atherosclerosis.
- CD36 mediates the phagocytosis of Plasmodium falciparum-infected erythrocytes by rodent macrophages.
- Annexin II regulates fibrin homeostasis and neoangiogenesis in vivo.
- CD36: a critical anti-angiogenic receptor.
- Kaposi's sarcoma associated herpesvirus G protein-coupled receptor immortalizes human endothelial cells by activation of the VEGF receptor-2/ KDR.
- CD36-mediated nonopsonic phagocytosis of erythrocytes infected with stage I and IIA gametocytes of Plasmodium falciparum.
- The face of TSR revealed: an extracellular signaling domain is exposed.
- A novel family of atherogenic oxidized phospholipids promotes macrophage foam cell formation via the scavenger receptor CD36 and is enriched in atherosclerotic lesions.
- Hematology Grants Workshop-2000.
Schools
Emory University School Of Medicine
Procedures Preformed
- Thoracentesis
Conditions Treated
- Anemia
Doctors Specialties
- Hematology
- Medical Oncology
- Oncology
Accepted Insurances
Awards
- Member
Education
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New York Presbyterian Hospital - Cornell Campus -
Emory University School of Medicine
Hospital
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Froedtert Memorial Lutheran Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1205892379 |
| Last Name Of The Provider | SILVERSTEIN |
| First Name Of The Provider | ROY |
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