Physician
Dermatology
Dr. Mina Yaar, MD
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Mina Yaar practices Dermatology. She studied medicine at The Hebrew University of Jerusalem Hadassah Medical School. She received awards:"CMS Meaningful Use Stage 1 Certification", "Fellow (FAAD)" and "Super Doctor". Mina Yaar is a published doctor as well. She has 101 publications published. The latest was: Thymidine dinucleotide mimics the effect of solar simulated irradiation on p53 and p53-regulated proteins. She is registered with Medicare and accepts Medicare payments.
Publications
- Cutaneous pigmentation in health and Disease: novel and well-established players.
- Melanocytes: a window into the nervous system.
- Inhibition of melanoma angiogenesis by telomere homolog oligonucleotides.
- Role of BMP-4 and Its Signaling Pathways in Cultured Human Melanocytes.
- Telomeric DNA induces p53-dependent reactive oxygen species and protects against oxidative damage.
- Keratin 16 expression in epidermal melanocytes.
- Telomere-mediated effects on melanogenesis and Skin aging.
- Cellular mechanisms regulating human melanogenesis.
- p75NTR antagonistic cyclic peptide decreases the size of beta amyloid-induced brain inflammation.
- Lymphangiogenesis and angiogenesis in non-phymatous Rosacea.
- Photoageing: mechanism, prevention and therapy.
- A cyclic peptide that binds p75(NTR) protects neurones from beta amyloid (1-40)-induced Cell death.
- Cutaneous immunoreactivity of D2-40 antibody beyond the lymphatics.
- Telomeric DNA induces apoptosis and senescence of human breast carcinoma Cells.
- Features that determine telomere homolog oligonucleotide-induced therapeutic DNA damage-like responses in Cancer cells.
- Neurotrophins in Skin biology and pathology.
- Bone morphogenetic protein-4, a novel modulator of melanogenesis.
- Keratin 16 expression in epidermal melanocytes of normal human Skin.
- Modulation of vascular endothelial growth factor receptors in melanocytes.
- [Advances in Skin aging/photoaging: theoretical and practical implications].
- Estradiol induces proliferation of keratinocytes via a receptor mediated mechanism.
- Melanocyte biology: before, during, and after the Fitzpatrick era.
- Solar-simulated irradiation evokes a persistent and biphasic IL-1alpha response.
- A trial of short incubation, broad-area photodynamic therapy for facial Actinic keratoses and diffuse photodamage.
- Modulations of nerve growth factor and Bcl-2 in ultraviolet-irradiated human epidermis.
- UV induces VEGF through a TNF-alpha independent pathway.
- p75 Neurotrophin receptor antagonist retards apoptosis-driven Hair follicle involution (catagen).
- Fifty years of Skin aging.
- Effect of sunscreen application on UV-induced thymine dimers.
- Mechanisms of aging.
- Tyrosinase gene expression is regulated by p53.
- Amyloid beta binds trimers as well as monomers of the 75-kDa neurotrophin receptor and activates receptor signaling.
- Ageing and photoageing of keratinocytes and melanocytes.
- Skin aging: postulated mechanisms and consequent changes in structure and function.
- Solar simulated irradiation modulates gene expression and activity of antioxidant enzymes in cultured human dermal fibroblasts.
- Vitiligo: the evolution of cultured epidermal autografts and other surgical treatment modalities.
- Effect of daily versus intermittent sunscreen application on solar simulated UV radiation-induced Skin response in humans.
- Mechanisms and implications of the age-associated decrease in DNA repair capacity.
- Role of cytoplasmic dynein in melanosome transport in human melanocytes.
- Kinesin participates in melanosomal movement along melanocyte dendrites.
- Thymidine dinucleotides inhibit contact hypersensitivity and activate the gene for tumor necrosis factor alpha1.
- The pathogenesis of melanoma induced by ultraviolet radiation.
- Thymidine dinucleotide mimics the effect of solar simulated irradiation on p53 and p53-regulated proteins.
- Aging versus photoaging: postulated mechanisms and effectors.
- Post-transcriptional regulation of UV induced TNF-alpha expression.
- Nerve growth factor: its significance in cutaneous biology.
- Purpuric drug eruption secondary to itraconazole.
- Binding of beta-amyloid to the p75 neurotrophin receptor induces apoptosis. A possible mechanism for Alzheimer's Disease.
- Human melanocytes as a model system for studies of Alzheimer Disease.
- Fas/Fas ligand interaction contributes to UV-induced apoptosis in human keratinocytes.
- Skin aging: observations at the cellular and molecular levels.
- Innervation of melanocytes in human Skin.
- Aging affects epidermal growth factor receptor phosphorylation and traffic kinetics.
- p75 nerve growth factor receptor staining helps identify desmoplastic and neurotropic melanoma.
- Proopiomelanocortin gene product regulation in keratinocytes.
- Nerve growth factor rescues pigment Cells from ultraviolet-induced apoptosis by upregulating BCL-2 levels.
- Mechanisms of ultraviolet light-induced pigmentation.
- Gene modulation accompanying differentiation of normal versus malignant keratinocytes.
- Influence of aging and malignant transformation on keratinocyte gene expression.
- Aging and photoaging affect gene expression in cultured human keratinocytes.
- Treatment of human melanocytes and S91 melanoma Cells with the DNA repair enzyme T4 endonuclease V enhances melanogenesis after ultraviolet irradiation.
- Effect of donor age on epidermal growth factor processing in man.
- E-cadherin is the major mediator of human melanocyte adhesion to keratinocytes in vitro.
- DNA damage and melanogenesis.
- The trk family of receptors mediates nerve growth factor and neurotrophin-3 effects in melanocytes.
- Role of integrins in melanocyte attachment and dendricity.
- Molecular mechanisms of Skin aging.
- Keratinocytes and fibroblasts in a human Skin equivalent model enhance melanocyte survival and melanin synthesis after ultraviolet irradiation.
- In vivo and in vitro SPRR1 gene expression in normal and malignant keratinocytes.
- A role for interleukin-1 in epidermal differentiation: regulation by expression of functional versus decoy receptors.
- Endothelin-1 of keratinocyte origin is a mediator of melanocyte dendricity.
- Retinoic acid delays the terminal differentiation of keratinocytes in suspension culture.
- Long-lasting cerebral functional changes following moderate dose x-radiation treatment to the scalp in childhood: an electroencephalographic power spectral study.
- Pemphigus antibodies identify a Cell surface glycoprotein synthesized by human and mouse keratinocytes.
- Laminin and bullous pemphigoid antigen are distinct basement membrane proteins synthesized by epidermal Cells.
- Immunopathological and clinical studies in herpes gestationis.
- The Goodpasture-like Syndrome in mice induced by intravenous injections of anti-type IV collagen and anti-laminin antibody.
- Abortion in mice induced by intravenous injections of antibodies to type IV collagen or laminin.
- Distribution and immunoelectron microscopic localization of laminin, a noncollagenous basement membrane glycoprotein.
- Stimulation of retinoic acid of synthesis and turnover of basement membrane in mouse embryonal carcinoma-derived endoderm Cells.
- Effects of alpha and beta interferons on cultured human keratinocytes.
- Substrate influences human epidermal melanocyte attachment and spreading in vitro.
- Normal human epidermis contains an interferon-like protein.
- Characteristics of cultivated adult human nevoCellular nevus Cells.
- Bullous pemphigoid: Disease of the aging immune system.
- Human nevoCellular nevus Cells are surrounded by basement membrane components. Immunohistologic studies of human nevus Cells and melanocytes in vivo and in vitro.
- Induction of nerve growth factor receptors on cultured human melanocytes.
- Interferon and the epidermis: implications for Cellular senescence.
- Normal human keratinocytes contain an interferon-like protein that may modulate their growth and differentiation.
- An extract of bovine thymus stimulates human keratinocyte growth in vitro.
- Cellular and molecular mechanisms of cutaneous aging.
- Dissociation of proto-oncogene induction from growth response in normal human fibroblasts.
- Oncogenes. The growth control genes.
- Human melanocyte growth and differentiation: a decade of new data.
- Variable keratin polypeptide profile in human stratum corneum.
- Immediate and delayed molecular response of human keratinocytes to solar-simulated irradiation.
- Evidence for nerve growth factor-mediated paracrine effects in human epidermis.
- Effect of aging and habitual sun exposure on the genetic response of cultured human keratinocytes to solar-simulated irradiation.
- Ageing and photoageing of the Skin: observations at the cellular and molecular level.
- Evidence that filopodia outgrowth is a common final pathway for fibroblast growth inhibition in vitro.
Schools
The Hebrew University Hadassah Medical School
Hadassah Med Center
Hadassah Hospital
New Eng Med Center Boston University
Tufts University
Procedures Preformed
- Destruction of Benign Skin Lesion
Conditions Treated
- Actinic Keratosis
- Athlete's Foot
- Benign Skin Lesion
- Dermatitis
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Doctors Specialties
- Dermatology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Fellow (FAAD)
- Super Doctor
Education
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Boston University Medical Center -
The Hebrew University of Jerusalem Hadassah Medical School
Hospital
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Boston Medical Center
Drug Facts
| NPI NUMBER | 1457342495 | |
| NPPES Provider LastName | YAAR | |
| NPPES Provider FirstName | MINA | |
| NPPES Provider ZIPCode | 020623155 | |
| NPPES Provider State | MA | |
| Specialty Description | Dermatology | |
| Total Claim Count | 211.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1457342495 |
| Last Name Of The Provider | YAAR |
| First Name Of The Provider | MINA |
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