Associate Professor at Stanford/VA Palo Alto Chief, PCCM
Pulmonology
Dr. Mark R. Nicolls, MD
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Mark Nicolls works in the field of Pulmonology. He graduated from Stanford University School of Medicine. Stanford University School of Medicine is a top medical university. Stanford University School of Medicine is ranked 2/25 in Research/PrimaryCare. He received awards:"CMS Meaningful Use Stage 1 Certification". Dr. Mark R. Nicolls is a published doctor as well. He has 46 publications published. The latest was: Microvascular injury after lung transplantation. Mark Nicolls accepts Medicare payments and is listed with Medicare.gov.
Publications
- Leukotriene B4 Activates Pulmonary Artery Adventitial Fibroblasts in Pulmonary Hypertension.
- Noninvasive monitoring of infection and rejection after Lung transplantation.
- Activation of the Wnt/Planar Cell Polarity Pathway Is Required for Pericyte Recruitment during Pulmonary Angiogenesis.
- Increased Resource Utilization in Lung Transplant Admissions in the Lung Allocation Score Era.
- Graft microvascular Disease in solid organ transplantation.
- NF-κB inhibition reduces lung vascular lumen obliteration in severe Pulmonary hypertension in rats.
- Leukotrienes in Pulmonary arterial hypertension.
- Promotion of airway anastomotic microvascular regeneration and alleviation of airway ischemia by deferoxamine nanoparticles.
- Temporal response of the human virome to immunosuppression and antiviral therapy.
- Application of a non-amplification-based technology to detect invasive fungal pathogens.
- The Critical Role of mRNA Destabilizing Protein Heterogeneous Nuclear Ribonucleoprotein D in 3' Untranslated Region-Mediated Decay of Low-Density Lipoprotein Receptor ...
- Efficacy of Transthoracic Echocardiography for Diagnosing Heart Failure in Septic Shock.
- Blocking macrophage leukotriene b4 prevents endothelial injury and reverses Pulmonary hypertension.
- Targeting complement component 5a promotes vascular integrity and limits airway remodeling.
- Complement-mediated microvascular injury leads to Chronic rejection.
- Developing better biomarkers for connective tissue Disease-associated interstitial lung Disease: Citrullinated HSP-90 autoantibodies in rheumatoid arthritis.
- Pathobiology of Pulmonary arterial hypertension and right ventricular failure.
- Potential for overuse of corticosteroids and vasopressin in septic shock.
- CD4+ T Cells and Complement Independently Mediate Graft Ischemia in the Rejection of Mouse Orthotopic Tracheal Transplants.
- Regulatory T Cells Limit Vascular Endothelial Injury and Prevent Pulmonary Hypertension.
- Regulatory T cells and Pulmonary hypertension.
- Adenovirus-mediated HIF-1α gene transfer promotes repair of mouse airway allograft microvasculature and attenuates Chronic rejection.
- Inhibiting Lung Elastase Activity Enables Lung Growth in Mechanically Ventilated Newborn Mice.
- Characterization of connective tissue disease-associated Pulmonary arterial hypertension from REVEAL: identifying systemic sclerosis as a unique phenotype.
- Bronchial blood supply after Lung transplantation without bronchial artery revascularization.
- Lung transplant airway hypoxia: a diathesis to Fibrosis?
- Microvascular destruction identifies murine allografts that cannot be rescued from airway Fibrosis.
- Oxidant stress, immune dysregulation, and vascular function in type I diabetes.
- Absence of T cells confers increased Pulmonary arterial hypertension and vascular remodeling.
- Hypoxia and the Lung: beyond hypoxic vasoconstriction.
- Transfer of allograft specific tolerance requires CD4+CD25+T cells but not interleukin-4 or transforming growth factor-beta and cannot induce tolerance to linked antig...
- The matrix comes to Lung transplantation.
- Is alveolar destruction and emphysema in Chronic obstructive pulmonary disease an immune disease?
- Mechanisms of autoimmune emphysema.
- The protective role of T-lymphocytes in Pulmonary vascular remodeling.
- Chemokine-mediated angiogenesis: an essential link in the evolution of airway Fibrosis?
- An animal model of autoimmune emphysema.
- Simultaneous LFA-1 and CD40 ligand antagonism prevents airway remodeling in orthotopic airway transplantation: implications for the role of respiratory epithelium as a...
- Gene microarray study corroborates proteomic findings in rodent islet cells.
- Proteomics as a tool for discovery: proteins implicated in Alzheimer's Disease are highly expressed in normal pancreatic islets.
- CD4-dependent generation of dominant transplantation tolerance induced by simultaneous perturbation of CD154 and LFA-1 pathways.
- Interferon-gamma is not a universal requirement for islet allograft survival.
- Macrophages in solid organ transplantation.
- Aspergillus fumigatus Invasion Increases with Progressive Airway Ischemia.
- Microvascular injury after Lung transplantation.
Schools
Stanford University
Procedures Preformed
- Bronchoscopy
- Thoracentesis
Conditions Treated
- Acidosis
- Asthma
- Bronchitis
- COPD (Chronic Obstructive Pulmonary Disease)
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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University of Colorado -
Stanford University School of Medicine
Hospital
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Stanford Health Care -
VA Palo Alto Health Care System
Drug Facts
| NPI NUMBER | 1497845176 | |
| NPPES Provider LastName | NICOLLS | |
| NPPES Provider FirstName | MARK | |
| NPPES Provider ZIPCode | 943055236 | |
| NPPES Provider State | CA | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 82.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1497845176 |
| Last Name Of The Provider | NICOLLS |
| First Name Of The Provider | MARK |
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