Physician
Internal Medicine
Dr. Mark P. Hoth, MD
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Mark Hoth works in the field of Internal Medicine. He went to St. George's University School of Medicine. Mark Hoth is a published doctor and has 43 publications published. The lastest was: 'SNARE protein expression and localization in human cytotoxic T lymphocytes.' Doctor Mark P. Hoth, MD accepts Medicare payments and is listed with Medicare.gov.
Publications
- Ion channels in health and disease. 83rd Boehringer Ingelheim Fonds International Titisee Conference.
- Mitochondrial control of calcium-channel gating: a mechanism for sustained signaling and transcriptional activation in T lymphocytes.
- TRP4 (CCE1) protein is part of native calcium release-activated Ca2+-like channels in adrenal cells.
- Absence of the gamma subunit of the skeletal muscle dihydropyridine receptor increases L-type Ca2+ currents and alters channel inactivation properties.
- TRP channels in lymphocytes.
- TRPC3 mediates T-cell receptor-dependent calcium entry in human T-lymphocytes.
- The PDZ-interacting domain of TRPC4 controls its localization and surface expression in HEK293 cells.
- Enhancement of calcium signalling dynamics and stability by delayed modulation of the plasma-membrane calcium-ATPase in human T cells.
- Skeletal muscle L-type Ca(2+) current modulation in gamma1-deficient and wildtype murine myotubes by the gamma1 subunit and cAMP.
- Sustained activity of calcium release-activated calcium channels requires translocation of mitochondria to the plasma membrane.
- TRPV6 potentiates calcium-dependent cell proliferation.
- Calcium-dependent activation of T-lymphocytes.
- Two-photon analysis of calcium signals in T lymphocytes of intact lamina propria from human intestine.
- Apparent cytosolic calcium gradients in T-lymphocytes due to fura-2 accumulation in mitochondria.
- Potent inhibition of Ca2+ release-activated Ca2+ channels and T-lymphocyte activation by the pyrazole derivative BTP2.
- Ca2+ signaling in identified T-lymphocytes from human intestinal mucosa. Relation to hyporeactivity, proliferation, and inflammatory bowel disease.
- SNARE protein expression and localization in human cytotoxic T lymphocytes.
- Pharmacology of ORAI channels as a tool to understand their physiological functions.
- Excitable T cells: Ca(v)1.4 channel contributions and controversies.
- Redox regulation of calcium ion channels: chemical and physiological aspects.
- Calcium microdomains at the immunological synapse: how ORAI channels, mitochondria and calcium pumps generate local calcium signals for efficient T-cell activation.
- ORAI-mediated calcium influx in T cell proliferation, apoptosis and tolerance.
- Docking of lytic granules at the immunological synapse in human CTL requires Vti1b-dependent pairing with CD3 endosomes.
- Calcium binding and transport by coenzyme Q.
- Immune synapses: mitochondrial morphology matters.
- Syntaxin7 is required for lytic granule release from cytotoxic T lymphocytes.
- Nonsteroidal anti-inflammatory drugs inhibit vascular smooth muscle cell proliferation by enabling the Ca2+-dependent inactivation of calcium release-activated calcium...
- TRP expression pattern and the functional importance of TRPC3 in primary human T-cells.
- Thapsigargin induces expression of activating transcription factor 3 in human keratinocytes involving Ca2+ ions and c-Jun N-terminal protein kinase.
- Differential redox regulation of ORAI ion channels: a mechanism to tune cellular calcium signaling.
- Mitochondria positioning controls local calcium influx in T cells.
- Efficiency of T-cell costimulation by CD80 and CD86 cross-linking correlates with calcium entry.
- The immunological synapse controls local and global calcium signals in T lymphocytes.
- A new rapid and simple method to determine the kinetics of electrode reactions of biologically relevant compounds from the half-peak width of the square-wave voltammog...
- Morphological changes of T cells following formation of the immunological synapse modulate intracellular calcium signals.
- Protein-film voltammetry: a theoretical study of the temperature effect using square-wave voltammetry.
- Probing the redox activity of T-lymphocytes deposited at electrode surfaces with voltammetric methods.
- Redox properties of the calcium chelator Fura-2 in mimetic biomembranes.
- Calcium dependence of T cell proliferation following focal stimulation.
- T cell activation requires mitochondrial translocation to the immunological synapse.
- Disruption of the cortical actin cytoskeleton does not affect store operated Ca2+ channels in human T-cells.
- P2X(7) receptor stimulation upregulates Egr-1 biosynthesis involving a cytosolic Ca(2+) rise, transactivation of the EGF receptor and phosphorylation of ERK and Elk-1.
Doctors Specialties
- Internal Medicine
Accepted Insurances
Education
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St. George's University School of Medicine
Drug Facts
| NPI NUMBER | 1083925606 | |
| NPPES Provider LastName | HOTH | |
| NPPES Provider FirstName | MARK | |
| NPPES Provider ZIPCode | 847902123 | |
| NPPES Provider State | UT | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 427.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1083925606 |
| Last Name Of The Provider | HOTH |
| First Name Of The Provider | MARK |
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