Physician
Pulmonology
Dr. John J. Osterholzer, MD
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John Osterholzer works in the field of Pulmonology. He attended University of Michigan Medical School. John Osterholzer is a published doctor and has 22 publications published. The lastest was: 'Local GM-CSF-Dependent Differentiation and Activation of Pulmonary Dendritic Cells and Macrophages Protect against Progressive Cryptococcal Lung Infection in Mice.'
Publications
- Local GM-CSF-Dependent Differentiation and Activation of Pulmonary Dendritic Cells and Macrophages Protect against Progressive Cryptococcal Lung Infection in Mice.
- Cryptococcus neoformans-Induced Macrophage Lysosome Damage Crucially Contributes to Fungal Virulence.
- Resident Alveolar Macrophages Suppress, whereas Recruited Monocytes Promote, Allergic Lung Inflammation in Murine Models of Asthma.
- Early or Late IL-10 Blockade Enhances Th1 and Th17 Effector Responses and Promotes Fungal Clearance in Mice with Cryptococcal Lung Infection.
- IL-17A enhances host defense against cryptococcal Lung infection through effects mediated by leukocyte recruitment, activation, and IFN-γ production.
- Scavenger receptor A modulates the immune response to Pulmonary Cryptococcus neoformans infection.
- Implicating Exudate Macrophages and Ly-6Chigh Monocytes in CCR2-Dependent Lung Fibrosis following Gene-Targeted Alveolar Injury.
- Virulence Factors Identified by Cryptococcus neoformans Mutant Screen Differentially Modulate Lung Immune Responses and Brain Dissemination.
- Macrophage Dectin-1 Expression Is Controlled by Leukotriene B4 via a GM-CSF/PU.1 Axis.
- PAI-1 Promotes the Accumulation of Exudate Macrophages and Worsens Pulmonary Fibrosis Following Type II Alveolar Epithelial Cell Injury.
- Coevolution of TH1, TH2, and TH17 responses during repeated Pulmonary exposure to Aspergillus fumigatus conidia.
- Chemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal Lung infection.
- Dual roles of CD40 on microbial containment and the development of immunopathology in response to persistent fungal infection in the Lung.
- Accumulation of CD11b+ Lung dendritic cells in response to fungal infection results from the CCR2-mediated recruitment and differentiation of Ly-6Chigh monocytes.
- Th2 but not Th1 immune bias results in altered Lung functions in a murine model of pulmonary Cryptococcus neoformans infection.
- Role of dendritic cells and alveolar macrophages in regulating early host defense against Pulmonary infection with Cryptococcus neoformans.
- Cryptococcal urease promotes the accumulation of immature dendritic cells and a non-protective T2 immune response within the Lung.
- Tyro3 receptor tyrosine kinases in the heterogeneity of apoptotic cell uptake.
- CCR2 mediates conventional dendritic cell recruitment and the formation of bronchovascular mononuclear cell infiltrates in the Lungs of mice infected with Cryptococcus...
- TLR9 is required for protective innate immunity in Gram-negative bacterial pneumonia: role of dendritic cells.
- CCR2 and CCR6, but not endothelial selectins, mediate the accumulation of immature dendritic cells within the Lungs of mice in response to particulate antigen.
Schools
University Of Michigan Medical School
University Of Mi Hosps and Health Ctrs
Conditions Treated
- Lung Cancer
- Pulmonary Hypertension
- Sleep Apnea
Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Education
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University of Michigan Hospitals and Health Centers -
University of Michigan Medical School
Hospital
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University of Michigan Hospitals and Health Centers
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