Professor, Medicine, UCSF School of Medicine
Infectious Disease
Dr. Joanne N. Engel, MD
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Dr. Joanne N. Engel is an expert in the field of Infectious Disease. She graduated from Stanford University School of Medicine. Stanford University School of Medicine is a top medical college. Stanford University School of Medicine is ranked 2 in research and 25 in primary care medicine She received awards:"Elected Member". Joanne N. Engel, MD is a published MD as well. She has 55 publications published. The latest was: Mutational analysis of the Chlamydia trachomatis rRNA P1 promoter defines four regions important for transcription in vitro.
Publications
- The Pseudomonas aeruginosa Type III Translocon Is Required for Biofilm Formation at the Epithelial Barrier.
- Host Cell Polarity Proteins Participate in Innate Immunity to Pseudomonas aeruginosa Infection.
- Sugar administration is an effective adjunctive therapy in the treatment of Pseudomonas aeruginosa pneumonia.
- Endosulfatases SULF1 and SULF2 limit Chlamydia muridarum infection.
- The Second Type VI Secretion System of Pseudomonas aeruginosa Strain PAO1 Is Regulated by Quorum Sensing and Fur and Modulates Internalization in Epithelial Cells.
- Pseudomonas aeruginosa Pili and Flagella Mediate Distinct Binding and Signaling Events at the Apical and Basolateral Surface of Airway Epithelium.
- Lipid acquisition by intracellular Chlamydiae.
- Chlamydia trachomatis Co-opts the FGF2 Signaling Pathway to Enhance Infection.
- Chlamydia trachomatis Co-opts GBF1 and CERT to Acquire Host Sphingomyelin for Distinct Roles during Intracellular Development.
- FimL regulates cAMP synthesis in Pseudomonas aeruginosa.
- Species-specific interactions of SRC family tyrosine kinases regulate Chlamydia intracellular growth and trafficking.
- Pseudomonas aeruginosa-mediated damage requires distinct receptors at the apical and basolateral surfaces of the polarized epithelium.
- Genetic analysis of the regulation of type IV pilus function by the Chp chemosensory system of Pseudomonas aeruginosa.
- CbpA: a polarly localized novel cyclic AMP-binding protein in Pseudomonas aeruginosa.
- RNAi screen reveals an Abl kinase-dependent host cell pathway involved in Pseudomonas aeruginosa internalization.
- RNA interference screen identifies Abl kinase and PDGFR signaling in Chlamydia trachomatis entry.
- Epithelial cell polarity alters Rho-GTPase responses to Pseudomonas aeruginosa.
- Modulation of cytosolic Ca(2+) concentration in airway epithelial cells by Pseudomonas aeruginosa.
- Type III protein secretion is associated with poor clinical outcomes in patients with ventilator-associated pneumonia caused by Pseudomonas aeruginosa.
- Assessing pseudomonas virulence using host cells.
- The arginine finger domain of ExoT contributes to actin cytoskeleton disruption and inhibition of internalization of Pseudomonas aeruginosa by epithelial cells and mac...
- Pseudomonas aeruginosa ExoT acts in vivo as a GTPase-activating protein for RhoA, Rac1, and Cdc42.
- The phosphoinositol-3-kinase-protein kinase B/Akt pathway is critical for Pseudomonas aeruginosa strain PAK internalization.
- Pseudomonas aeruginosa induces type-III-secretion-mediated apoptosis of macrophages and epithelial cells.
- Defects in type III secretion correlate with internalization of Pseudomonas aeruginosa by epithelial cells.
- Pili binding to asialo-GM1 on epithelial cells can mediate cytotoxicity or bacterial internalization by Pseudomonas aeruginosa.
- Pseudomonas aeruginosa gene products PilT and PilU are required for cytotoxicity in vitro and virulence in a mouse model of acute pneumonia.
- Rho GTPase activity modulates Pseudomonas aeruginosa internalization by epithelial cells.
- Pseudomonas aeruginosa fimL regulates multiple virulence functions by intersecting with Vfr-modulated pathways.
- Identification of Pseudomonas aeruginosa genes required for epithelial cell injury.
- PepA, a secreted protein of Pseudomonas aeruginosa, is necessary for cytotoxicity and virulence.
- Host cell-derived sphingolipids are required for the intracellular growth of Chlamydia trachomatis.
- Pseudomonas aeruginosa ExoT inhibits in vitro lung epithelial wound repair.
- Infection with Chlamydia trachomatis alters the tyrosine phosphorylation and/or localization of several host cell proteins including cortactin.
- Cloning and characterization of RNA polymerase core subunits of Chlamydia trachomatis by using the polymerase chain reaction.
- Eukaryotic cell uptake of heparin-coated microspheres: a model of host cell invasion by Chlamydia trachomatis.
- A polymerase chain reaction-based approach to cloning sigma factors from eubacteria and its application to the isolation of a sigma-70 homolog from Chlamydia trachomatis.
- Transcriptional organization and regulation of the dnaK and groE operons of Chlamydia trachomatis.
- Drosophila melanogaster S2 cells: a model system to study Chlamydia interaction with host cells.
- Characterization of a complex chemosensory signal transduction system which controls twitching motility in Pseudomonas aeruginosa.
- Roles of type IV pili, flagellum-mediated motility and extracellular DNA in the formation of mature multicellular structures in Pseudomonas aeruginosa biofilms.
- Lipid raft-mediated entry is not required for Chlamydia trachomatis infection of cultured epithelial cells.
- The ADP ribosyltransferase domain of Pseudomonas aeruginosa ExoT contributes to its biological activities.
- Heat shock response of murine Chlamydia trachomatis.
- Identification of sequences necessary for transcription in vitro from the Chlamydia trachomatis rRNA P1 promoter.
- Mutational analysis of the Chlamydia trachomatis rRNA P1 promoter defines four regions important for transcription in vitro.
- Interaction of bacterial pathogens with polarized epithelium.
- A mycobacterial virulence gene cluster extending RD1 is required for cytolysis, bacterial spreading and ESAT-6 secretion.
- Chlamydial rRNA operons: gene organization and identification of putative tandem promoters.
- Chlamydial gene encoding a 70-kilodalton antigen in Escherichia coli: analysis of expression signals and identification of the gene product.
- Azithromycin-induced block of elementary body formation in Chlamydia trachomatis.
- A developmentally regulated chlamydial gene with apparent homology to eukaryotic histone H1.
- Stage-specific regulation of protein phosphorylation in Leishmania major.
- Chlamydial intracellular survival strategies.
Schools
Stanford University School Of Medicine
University Of Pennsylvania
Hospital Of The University Of Pennsylvania
University Calif
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Awards
- Elected Member
Education
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University of Pennsylvania Health System -
Stanford University School of Medicine
Hospital
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San Francisco General Hospital and Trauma Center -
UCSF Medical Center
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