Professor, Medicine, Vanderbilt University School of Medicine
Pulmonology
Dr. James Sheller, MD
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James Sheller works in the field of Pulmonology. He went to Vanderbilt University School of Medicine. He over the years received 1 awards: "Meaningful Use Stage 1 Certification". James Sheller, MD is a published doctor as well. He has 93 publications published. The latest was: Respiratory syncytial virus infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin-sensitized mice. He accepts Medicare.
Publications
- Pharmacogenetic determinants of codeine induction by rifampin: the impact on codeine's Respiratory, psychomotor and miotic effects.
- Pharmacogenetic determination of the effects of codeine and prediction of drug interactions.
- Effect of leukotrienes on sheep airway smooth muscle.
- Impact of ethnic origin and quinidine coadministration on codeine's disposition and pharmacodynamic effects.
- A retrospective characterization of African- and European American Asthmatic children in a pediatric critical care unit.
- The prostaglandin E agonist, misoprostol, inhibits airway IL-5 production in atopic Asthmatics.
- Localization of isoketal adducts in vivo using a single-chain antibody.
- Biochemical and clinical evidence that aspirin-intolerant Asthmatic subjects tolerate the cyclooxygenase 2-selective analgetic drug celecoxib.
- Insights into IgE-mediated Lung inflammation derived from a study employing a 5-lipoxygenase inhibitor.
- Inhaled steroids in Asthma.
- Bronchial aspirin challenge causes specific eicosanoid response in aspirin-sensitive Asthmatics.
- Bronchomotor responses of isolated sheep airways to electrical field stimulation.
- Barbiturates depress vagal motor pathway to ferret trachea at ganglia.
- Interaction of serotonin with vagal- and ACh-induced bronchoconstriction in canine Lungs.
- Bronchial hyperreactivity.
- Threshold concentration of ozone causing an increase in bronchial reactivity in humans and adaptation with repeated exposures.
- Effect of ganglionic blockade on bronchial reactivity in atopic subjects.
- Arachidonic acid metabolites in the healthy and diseased Lung.
- Transbronchoscopic needle aspiration in the diagnosis and staging of Lung cancer.
- Acute effects of thoracic irradiation on Lung function and structure in awake sheep.
- CNS tuberculosis.
- (5Z,13E)-(15S)-9 alpha,11 beta,15-trihydroxyprosta-5,13-dien-1-oic acid (9 alpha,11 beta-prostaglandin F2): formation and metabolism by human Lung and contractile effe...
- Pulmonary function in long-term survivors of hyaline membrane disease.
- Prostaglandins in Ascaris antigen.
- Right ventricular hypertrophy is an important determinant of right ventricular infarction complicating acute inferior left ventricular infarction.
- 9 alpha, 11 beta-prostaglandin F2: formation and metabolism by human Lung and contractile effects on human bronchial smooth muscle.
- Asthma: emerging concepts and potential therapies.
- Postpneumonic Empyema in childhood: selecting appropriate therapy.
- Prematurity is associated with abnormal airway function in childhood.
- Airway responsiveness in isolated perfused rat Lungs: effect of thoracic irradiation.
- Leukotrienes and ARDS.
- Thromboxane biosynthesis in allergen-induced Bronchospasm. Evidence for platelet activation.
- Allergen-stimulated release of mediators into sheep bronchoalveolar lavage fluid. Effect of cyclooxygenase inhibition.
- A controlled trial of the effect of the 5-lipoxygenase inhibitor, zileuton, on Lung inflammation produced by segmental antigen challenge in human beings.
- Inhibition of eicosanoid biosynthesis by glucocorticoids in humans.
- Allergen-stimulated release of thromboxane A2 and leukotriene E4 in humans. Effect of indomethacin.
- Formation of PGD2 after allergen inhalation in atopic Asthmatics.
- Thromboxane synthase inhibition in allergen challenged sheep Lung: effect on eicosanoid synthesis.
- Effect of ozone on bronchial reactivity in atopic and nonatopic subjects.
- Ethnic differences in response to morphine.
- Effect of oral prednisone on airway inflammatory mediators in atopic Asthma.
- Eicosanoids in bronchoalveolar lavage fluid of aspirin-intolerant patients with Asthma after aspirin challenge.
- Role of leukotrienes revealed by targeted disruption of the 5-lipoxygenase gene.
- New therapies for Asthma.
- Regional distribution of protease-resistant prion protein in fatal familial insomnia.
- Immune interaction between Respiratory syncytial virus infection and allergen sensitization critically depends on timing of challenges.
- IL-4 signaling, gene transcription regulation, and the control of effector T cells.
- Assessment of oxidant stress in allergic Asthma by measurement of the major urinary metabolite of F2-isoprostane, 15-F2t-IsoP (8-iso-PGF2alpha).
- Respiratory syncytial virus infection does not increase allergen-induced type 2 cytokine production, yet increases airway hyperresponsiveness in mice.
- Induced sputum inflammatory mediator concentrations in eosinophilic bronchitis and Asthma.
- Respiratory syncytial virus (RSV)-induced airway hyperresponsiveness in allergically sensitized mice is inhibited by live RSV and exacerbated by formalin-inactivated RSV.
- Cyclooxygenase inhibition increases interleukin 5 and interleukin 13 production and airway hyperresponsiveness in allergic mice.
- Prostaglandin E(2) decreases allergen-stimulated release of prostaglandin D(2) in airways of subjects with Asthma.
- EP(2) receptor mediates bronchodilation by PGE(2) in mice.
- Sputum cysteinyl leukotrienes increase 24 hours after allergen inhalation in atopic Asthmatics.
- Allergen-induced synthesis of F(2)-isoprostanes in atopic Asthmatics. Evidence for oxidant stress.
- Induced sputum eicosanoid concentrations in Asthma.
- Preferential role for NF-kappa B/Rel signaling in the type 1 but not type 2 T cell-dependent immune response in vivo.
- 5-Lipoxygenase products are necessary for ovalbumin-induced airway responsiveness in mice.
- Differential sensitivities of human blood monocytes and alveolar macrophages to the inhibition of prostaglandin endoperoxide synthase-2 by interleukin-4.
- Prednisone increases PGH-synthase 2 in atopic humans in vivo.
- Use of pulse oximetry to recognize severity of airflow obstruction in Obstructive airway disease: correlation with pulsus paradoxus.
- Respiratory syncytial virus infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin-sensitized mice.
- Urinary mediators and Asthma.
- In vivo function of an interleukin 2 receptor beta chain (IL-2Rbeta)/IL-4Ralpha cytokine receptor chimera potentiates allergic airway Disease.
- Salmeterol prevents aspirin-induced attacks of Asthma and interferes with eicosanoid metabolism.
- Suppression of airway hyperresponsiveness induced by ovalbumin sensitisation and RSV infection with Y-27632, a Rho kinase inhibitor.
- Quantitative analysis of f(2) isoprostanes.
- Quantitative analysis of cyclooxygenase metabolites of arachidonic Acid.
- Constrictive Bronchiolitis in soldiers returning from Iraq and Afghanistan.
- The RAD score: a simple acute Asthma severity score compares favorably to more complex scores.
- Accessory muscle use in pediatric patients with acute Asthma exacerbations.
- Cyclooxygenase inhibition augments allergic inflammation through CD4-dependent, STAT6-independent mechanisms.
- Susceptibility to allergic Lung disease regulated by recall responses of dual-receptor memory T cells.
- Recall helper T cell response: T helper 1 cell-resistant allergic susceptibility without biasing uncommitted CD4 T cells.
- Multi-organ inflammation after hepatic cryoablation in BALB/c mice.
- Discovery of lipid peroxidation products formed in vivo with a substituted tetrahydrofuran ring (isofurans) that are favored by increased oxygen tension.
- Selective cyclooxygenase-1 and -2 inhibitors each increase allergic inflammation and airway hyperresponsiveness in mice.
- Nuclear factor kappa B induction in airway epithelium increases Lung inflammation in allergen-challenged mice.
- Genetic variants of GSNOR and ADRB2 influence response to albuterol in African-American children with severe Asthma.
- Effect of allergen challenge on the percentage of natural killer T cells in patients with atopic Asthma.
- The Shanghai Women's Asthma and Allergy Study: objectives, design, and recruitment results.
- Changes in urinary dinor dihydro F(2)-isoprostane metabolite concentrations, a marker of oxidative stress, during and following Asthma exacerbations.
- Airway epithelium controls Lung inflammation and injury through the NF-kappa B pathway.
- Oxidant stress modulates murine allergic airway responses.
- Release of free F2-isoprostanes from esterified phospholipids is catalyzed by intracellular and plasma platelet-activating factor acetylhydrolases.
- Allergen-induced airway hyperresponsiveness mediated by cyclooxygenase inhibition is not dependent on 5-lipoxygenase or IL-5, but is IL-13 dependent.
- Respiratory syncytial virus infection in the absence of STAT 1 results in airway dysfunction, airway mucus, and augmented IL-17 levels.
- Soluble CD23 and interleukin-1 receptor antagonist in human Asthmatics following antigen challenge.
- Viral infections in Asthma.
- Flow volume loops in the evaluation of upper airway obstruction.
- Childhood Pulmonary function following hyaline membrane disease.
Schools
Vanderbilt University
Uc San Francisco Hosps
Curi
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asbestosis
- Asthma
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Amerigroup (Wellpoint)
- Blue Card PPO
- Network S
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Coventry Health Care
- Coventry Health Care of Tennessee PPO
- GEHA
- Choice Care PPO
- Medicare MCR
- PHCS Network PPO
- POMCO Group
- Tricare
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
- WellCare
Awards
- Meaningful Use Stage 1 Certification
Education
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UCSF -
Vanderbilt University School of Medicine
Hospital
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Vanderbilt Hospital and Clinics
Drug Facts
| NPI NUMBER | 1538268974 | |
| NPPES Provider LastName | SHELLER | |
| NPPES Provider FirstName | JAMES | |
| NPPES Provider ZIPCode | 372320001 | |
| NPPES Provider State | TN | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 344.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1538268974 |
| Last Name Of The Provider | SHELLER |
| First Name Of The Provider | JAMES |
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