Physician
Pathology
Dr. Gary R. Pasternack, MD
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Gary R. Pasternack works in the field of Pathology. He graduated from Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is a one of the best medicals schools. Johns Hopkins University School of Medicine has a rank of 3 in research and a 29 in primary care Gary Pasternack is a published MD as well. He has 48 publications published. The latest was: Antibody-mediated suppression of grafted lymphoma. IV. Influence of time of tumor residency in vivo and tumor size upon the effectiveness of suppression by syngeneic a...
Publications
- Reduction of pp32 expression in poorly differentiated pancreatic ductal adenocarcinomas and intraductal papillary mucinous neoplasms with moderate dysplasia.
- A pp32-retinoblastoma protein complex modulates androgen receptor-mediated transcription and associates with components of the splicing machinery.
- Phosphorylated retinoblastoma protein complexes with pp32 and inhibits pp32-mediated apoptosis.
- The identification of phosphorylation sites of pp32 and biochemical purification of a cellular pp32-kinase.
- Identification of a functional mutation in pp32r1 (ANP32C).
- pp32 reduction induces differentiation of TSU-Pr1 cells.
- Immunolocalization of band 3 protein in normal and cystic fibrosis skin.
- Three distinct cell populations in rat kidney collecting duct.
- Immunocytochemical characterization of carbonic anhydrase-rich cells in the rat kidney collecting duct.
- Immunocytochemistry of band 3 protein in kidney and other tissues of control and cystic fibrosis patients.
- The immunodominant major histocompatibility complex class I-restricted antigen of a murine colon tumor derives from an endogenous retroviral gene product.
- Inhibition of fatty acid synthesis induces programmed cell death in human breast cancer cells.
- Inhibition of fatty acid synthesis delays disease progression in a xenograft model of ovarian cancer.
- Fatty acid synthase (FAS): a target for cytotoxic antimetabolites in HL60 promyelocytic leukemia cells.
- Fatty acid synthesis: a potential selective target for antineoplastic therapy.
- Simplified high-sensitivity sequencing of a major histocompatibility complex class I-associated immunoreactive peptide using matrix-assisted laser desorption/ionizatio...
- A novel M(r) 32,000 nuclear phosphoprotein is selectively expressed in cells competent for self-renewal.
- The structural basis of ankyrin function. II. Identification of two functional domains.
- Interactions between protein 4.1 and band 3. An alternative binding site for an element of the membrane skeleton.
- Identification and preliminary characterization of two related proliferation-associated nuclear phosphoproteins.
- Microscale, filtration-type binding assay for studying myosin-erythrocyte protein 4.1 interactions.
- Expression of haptoglobin-related protein in primary and metastatic breast cancers. A longitudinal study of 48 fatal tumors.
- Expression of oncogenic antigen 519 (OA-519) in prostate cancer is a potential prognostic indicator.
- Antibody-mediated suppression of grafted lymphoma. IV. Influence of time of tumor residency in vivo and tumor size upon the effectiveness of suppression by syngeneic a...
- Immunotherapy of cancer with antibody.
- Antibody-mediated suppression of tumor growth. I. Suppression by murine IgG1 isolated from alloantiserum.
- Antibody-mediated suppression of tumor growth. III. Molecular assay of murine IgG1 alloantibody required to cause tumor suppression in vivo.
- Antibody-mediated suppression of tumor growth. II. Macrophage and platelet cooperation with murine IgG1 isolated from alloantiserum.
- Tumor cell cytostasis by macrophages and antibody in vitro. II. Isolation and characterization of suppressed cells.
- Mechanisms of tumor immunity: the role of antibody and nonimmune effectors.
- Resistance of neoplasms to immunological destruction: role of a macrophage chemotaxis inhibitor.
- Tumor cell cytostasis by macrophages and antibody in vitro. I. Resolution into contact-dependent and contact-independent steps.
- Expression of pp32 gene family members in breast cancer.
- Tumor suppression and potentiation by manipulation of pp32 expression.
- In situ hybridization-theory and practice.
- Identification of sequences required for inhibition of oncogene-mediated transformation by pp32.
- Modulation of oncogenic potential by alternative gene use in human prostate cancer.
- Enzymes of the fatty acid synthesis pathway are highly expressed in in situ breast carcinoma.
- Fatty acid synthase expression in endometrial carcinoma: correlation with cell proliferation and hormone receptors.
- Novel nuclear phosphoprotein pp32 is highly expressed in intermediate- and high-grade prostate cancer.
- pp32 overexpression induces nuclear pleomorphism in rat prostatic carcinoma cells.
- Elevated expression of fatty acid synthase and fatty acid synthetic activity in colorectal neoplasia.
- Expression of fatty acid synthase is closely linked to proliferation and stromal decidualization in cycling endometrium.
- Structure of pp32, an acidic nuclear protein which inhibits oncogene-induced formation of transformed foci.
- Erythrocyte protein 4.1 binds and regulates myosin.
- Haptoglobin-related protein (Hpr) epitopes in breast cancer as a predictor of recurrence of the disease.
- Expression of haptoglobin-related protein and its potential role as a tumor antigen.
Schools
Johns Hopkins University School of Medicine
Doctors Specialties
- Anatomic Pathology
- Pathology
Accepted Insurances
Education
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Yale-New Haven Hospital -
Johns Hopkins University School of Medicine
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