Professor, Medicine - Endocrinology, Gerontology, and Metabolism, Stanford University Medical Center
Endocrinology
Dr. Fredric B. Kraemer, MD
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Fredric Kraemer works in the field of Endocrinology. He went to medical school at NYU School of Medicine. He received 2 awards: "Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". Fredric Kraemer is a published doctor as well. He has 54 publications published. The latest was: A Novel Role of Salt-Inducible Kinase 1 (SIK1) in the Post-Translational Regulation of Scavenger Receptor Class B Type 1 Activity. He accepts Medicare payments and is listed with Medicare.gov.
Publications
- A Novel Role of Salt-Inducible Kinase 1 (SIK1) in the Post-Translational Regulation of Scavenger Receptor Class B Type 1 Activity.
- Scavenger Receptor class B Type I (SR-BI): A versatile receptor with multiple functions and actions.
- Lipid droplet metabolism.
- HPA axis genetic variation, cortisol and psychosis in major depression.
- Regulation of Expression and Function of Scavenger Receptor Class B, Type I (SR-BI) by Na+/H+ Exchanger Regulatory Factors (NHERFs).
- Mechanisms of action of hormone-sensitive lipase in mouse Leydig cells: its role in the regulation of the steroidogenic acute regulatory protein.
- Fat-specific protein 27 modulates nuclear factor of activated T cells 5 and the cellular response to stress.
- Nordihydroguaiaretic acid improves metabolic dysregulation and aberrant hepatic Lipid metabolism in mice by both PPAR?-dependent and -independent pathways.
- The mineralocorticoid receptor agonist, fludrocortisone, differentially inhibits pituitary-adrenal activity in humans With psychotic major depression.
- MicroRNAs 125a and 455 Repress Lipoprotein-Supported Steroidogenesis by Targeting Scavenger Receptor Class B Type I in Steroidogenic Cells.
- HSL-knockout mouse testis exhibits class B scavenger receptor upregulation and disrupted Lipid raft microdomains.
- Cholesterol ester droplets and steroidogenesis.
- Ablation of Vimentin Results in Defective Steroidogenesis.
- Lipid droplet meets a mitochondrial protein to regulate adipocyte lipolysis.
- Evidence for protein-mediated fatty acid efflux by adipocytes.
- Characterization of age-related gene expression profiling in bone marrow and epididymal adipocytes.
- The role of Lipid droplets in metabolic disease in rodents and humans.
- Hormone-sensitive lipase-knockout mice maintain high bone density during aging.
- Hormone-sensitive lipase modulates adipose metabolism through PPARgamma.
- IL-17 regulates adipogenesis, glucose homeostasis, and obesity.
- Adipocytes decrease Runx2 expression in osteoblastic cells: roles of PPARγ and adiponectin.
- Strong induction of PCSK9 gene expression through HNF1alpha and SREBP2: mechanism for the resistance to LDL-cholesterol lowering effect of statins in dysLipidemic hams...
- Vimentin is a functional partner of hormone sensitive lipase and facilitates lipolysis.
- Functional interaction of hormone-sensitive lipase and perilipin in lipolysis.
- Effects of hormone-sensitive lipase-disruption on cardiac energy metabolism in response to fasting and refeeding.
- Identification of mRNA binding proteins that regulate the stability of LDL receptor mRNA through AU-rich elements.
- Cardiac overexpression of hormone-sensitive lipase inhibits myocardial steatosis and fibrosis in streptozotocin Diabetic mice.
- Transcriptional activation of hepatic ACSL3 and ACSL5 by oncostatin m reduces hypertriglyceridemia through enhanced beta-oxidation.
- Generation of novel adipocyte monolayer cultures from embryonic stem cells.
- The LDL receptor is not necessary for acute adRenal steroidogenesis in mouse adrenocortical cells.
- Control of adipose triglyceride lipase action by serine 517 of perilipin A globally regulates protein kinase A-stimulated lipolysis in adipocytes.
- The medicinal plant goldenseal is a natural LDL-lowering agent With multiple bioactive components and new action mechanisms.
- Perilipin promotes hormone-sensitive lipase-mediated adipocyte lipolysis via phosphorylation-dependent and -independent mechanisms.
- In vivo activities of cytokine oncostatin M in the regulation of plasma Lipid levels.
- Mutational analysis of the "regulatory module" of hormone-sensitive lipase.
- Physical association between the adipocyte fatty acid-binding protein and hormone-sensitive lipase: a fluorescence resonance energy transfer analysis.
- Absence of hormone-sensitive lipase inhibits obesity and adipogenesis in Lep ob/ob mice.
- Hormone-sensitive lipase is required for high-density lipoprotein cholesteryl ester-supported adRenal steroidogenesis.
- Overexpression of leptin in transgenic mice leads to decreased basal lipolysis, PKA activity, and perilipin levels.
- Lipase-selective functional domains of perilipin A differentially regulate constitutive and protein kinase A-stimulated lipolysis.
- Resistance to high-fat diet-induced obesity and altered expression of adipose-specific genes in HSL-deficient mice.
- Fatty acid-binding protein-hormone-sensitive lipase interaction. Fatty acid dependence on binding.
- Angiotensin II activates cholesterol ester hydrolase in bovine adRenal glomerulosa cells through phosphorylation mediated by p42/p44 mitogen-activated protein kinase.
- Interaction of hormone-sensitive lipase With steroidogenic acute regulatory protein: facilitation of cholesterol transfer in adrenal.
- Sterol-mediated regulation of hormone-sensitive lipase in 3T3-L1 adipocytes.
- Hormone-sensitive lipase: control of intracellular tri-(di-)acylglycerol and cholesteryl ester hydrolysis.
- Identification of Egr1 as the oncostatin M-induced transcription activator that binds to sterol-independent regulatory element of human LDL receptor promoter.
- Cardiac gene expression profile and Lipid accumulation in response to starvation.
- Effectiveness of Diabetes management: is improvement feasible?
- Modulation of hormone-sensitive lipase and protein kinase A-mediated lipolysis by perilipin A in an adenoviral reconstituted system.
- AdRenal neutral cholesteryl ester hydrolase: identification, subcellular distribution, and sex differences.
- Hormonal Regulation of MicroRNA Expression in Steroid Producing Cells of the Ovary, Testis and AdRenal Gland.
- Age-related modulation of the effects of obesity on gene expression profiles of mouse bone marrow and epididymal adipocytes.
Schools
New York University
Kings Co Dwnst Med Center
Stanford University
Conditions Treated
- Diabetes Type 1
- Diabetes Type 2
- Diabetes With Renal Manifestations
- Diabetic Retinopathy
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Doctors Specialties
- Endocrinology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- Advantage Preferred DirectAccess Plus PPO
- Blue Cross Blue Shield of California PPO
- Select PPO
- Access Plus Savenet
- Basic Exclusive PPO for Small Business
- Basic PPO
- CHAMPVA
- Great West Healthcare-Cigna PPO
- Commercial Insurance Company
- First Health (Coventry Health Care) PPO
- Covered California - Bronze PPO
- Health Net PPO
- Individual and Family PPO
- Medicare Advantage PFFS
- Medicare MCR
- Kaiser PHCS Network PPO
- MultiPlan PPO
- PHCS Network PPO
- Mutual of Omaha
- TriWest Champus
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- PacifiCare HMO
- United Healthcare
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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Stanford University -
SUNY Health Science Center at Brooklyn -
NYU School of Medicine
Hospital
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VA Palo Alto Health Care System -
Stanford Health Care
Drug Facts
| NPI NUMBER | 1780796037 | |
| NPPES Provider LastName | KRAEMER | |
| NPPES Provider FirstName | FREDRIC | |
| NPPES Provider ZIPCode | 943055102 | |
| NPPES Provider State | CA | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 272.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1780796037 |
| Last Name Of The Provider | KRAEMER |
| First Name Of The Provider | FREDRIC |
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