Professor of Medicine and Human Genetics, University of Chicago Medicine
Cardiology
Dr. Elizabeth M. McNally, MD
Dr. Elizabeth M. McNally works in the field of Cardiology. She graduated from Albert Einstein College of Medicine of Yeshiva University. She over the years received 3 awards: "CMS Meaningful Use Stage 1 Certification", "Elected Member" and "Super Doctor". Elizabeth M. McNally, MD is also published. She has 79 publications published. The lastest publication: "The genetic landscape of cardiomyopathy and its role in heart failure.'
Publications
- The genetic landscape of cardiomyopathy and its role in Heart failure.
- Excess SMAD signaling contributes to Heart and muscle dysfunction in muscular dystrophy.
- Myofiber-specific inhibition of TGFβ signaling protects skeletal muscle from injury and dystrophic Disease in mice.
- Targeted Analysis of Whole Genome Sequence Data to Diagnose Genetic Cardiomyopathy.
- Two Strikes and You're Out: Gene-Gene Mutation Interactions in HCM.
- GRAF1 promotes ferlin-dependent myoblast fusion.
- Cardiovascular genetics: paying individual dividends.
- P38α MAPK underlies muscular dystrophy and myofiber death through a Bax-dependent mechanism.
- Modifiers of Heart and muscle function: where genetics meets physiology.
- Annexin A6 modifies muscular dystrophy by mediating sarcolemmal repair.
- Abcc9 is required for the transition to oxidative metabolism in the newborn Heart.
- EHD1 mediates vesicle trafficking required for normal muscle growth and transverse tubule development.
- Supercomputing for the parallelization of whole genome analysis.
- Dysferlin and Myoferlin Regulate Transverse Tubule Formation and Glycerol Sensitivity.
- Modifying muscular dystrophy through transforming growth factor-?
- Genetic mutations and mechanisms in dilated Cardiomyopathy.
- LTBP4 genotype predicts age of ambulatory loss in duchenne muscular dystrophy.
- S100A12 Expression in Thoracic Aortic Aneurysm Is Associated With Increased Risk of Dissection and Perioperative Complications.
- Genetic pathways of vascular calcification.
- Genetics of vascular calcification.
- Vasodilation induced by oxygen/glucose deprivation is attenuated in cerebral arteries of SUR2 null mice.
- Gene expression, chromosome position and lamin A/C mutations.
- The emerging genetic landscape underlying cardiac conduction system function.
- Hydrogen sulfide dilates cerebral arterioles by activating smooth muscle cell plasma membrane KATP channels.
- Endocytic recycling proteins EHD1 and EHD2 interact with fer-1-like-5 (Fer1L5) and mediate myoblast fusion.
- Cardiac magnetic resonance of left ventricular trabeculation: the new normal.
- S100A12 in vascular smooth muscle accelerates vascular calcification in apolipoprotein E-null mice by activating an osteogenic gene regulatory program.
- Distinct genetic regions modify specific muscle groups in muscular dystrophy.
- Ferlin proteins in myoblast fusion and muscle growth.
- The mitochondrial bioenergetic phenotype for protection from cardiac ischemia in SUR2 mutant mice.
- Cardiac assessment in duchenne and becker muscular dystrophies.
- Cardiomyocyte sulfonylurea receptor 2-KATP channel mediates cardioprotection and ST segment elevation.
- Mechanisms of muscle weakness in muscular dystrophy.
- Myoferlin is required for insulin-like growth factor response and muscle growth.
- Ventricular Fibrillation following autologous intramyocardial cell therapy for inherited cardiomyopathy.
- Nesprins, but not sun proteins, switch isoforms at the nuclear envelope during muscle development.
- The genetics of dilated Cardiomyopathy.
- Familial dilated Cardiomyopathy caused by an alpha-tropomyosin mutation: the distinctive natural history of sarcomeric dilated Cardiomyopathy.
- S100A12 mediates aortic wall remodeling and aortic Aneurysm.
- Latent TGF-beta-binding protein 4 modifies muscular dystrophy in mice.
- Molecular identification and functional characterization of a mitochondrial sulfonylurea receptor 2 splice variant generated by intraexonic splicing.
- Genetic manipulation of dysferlin expression in skeletal muscle: novel insights into muscular dystrophy.
- Healing health care.
- Impaired exercise tolerance and skeletal muscle myopathy in sulfonylurea receptor-2 mutant mice.
- Sarcomere mutations in Cardiomyopathy with left ventricular hypertrabeculation.
- Setting the pace: Tbx3 and Tbx18 in cardiac conduction system development.
- Mechanisms of muscle degeneration, regeneration, and repair in the muscular dystrophies.
- A pilot study of a family history risk assessment tool for cardiovascular Disease.
- Long-term survival of transplanted stem cells in immunocompetent mice with muscular dystrophy.
- Sulfonylurea receptor-dependent and -independent pathways mediate vasodilation induced by ATP-sensitive K+ channel openers.
- The endocytic recycling protein EHD2 interacts with myoferlin to regulate myoblast fusion.
- Sarcomere mutations in cardiomyopathy, noncompaction, and the developing Heart.
- Myostatin blockade improves function but not histopathology in a murine model of limb-girdle muscular dystrophy 2C.
- Myoferlin regulates vascular endothelial growth factor receptor-2 stability and function.
- Genetic disruption of calcineurin improves skeletal muscle pathology and cardiac Disease in a mouse model of limb-girdle muscular dystrophy.
- Consequences of disrupting the dystrophin-sarcoglycan complex in cardiac and skeletal myopathy.
- Muscle Diseases: the muscular dystrophies.
- New approaches in the therapy of Cardiomyopathy in muscular dystrophy.
- Phospholamban R14 deletion results in late-onset, mild, hereditary dilated Cardiomyopathy.
- Age-dependent effect of myostatin blockade on Disease severity in a murine model of limb-girdle muscular dystrophy.
- Spontaneous Coronary vasospasm in KATP mutant mice arises from a smooth muscle-extrinsic process.
- Hypertrophic Cardiomyopathy: exercise and eat right.
- Bacillus anthracis edema toxin causes extensive tissue lesions and rapid lethality in mice.
- The interaction of Coronary tone and cardiac fibrosis.
- Nesprin-1alpha contributes to the targeting of mAKAP to the cardiac myocyte nuclear envelope.
- Transplanted hematopoietic stem cells demonstrate impaired sarcoglycan expression after engraftment into cardiac and skeletal muscle.
- Regenerating more than muscle in muscular dystrophy.
- Powerful genes--myostatin regulation of human muscle mass.
- The dystrophin glycoprotein complex: signaling strength and integrity for the sarcolemma.
- Smooth muscle cell-extrinsic vascular spasm arises from cardiomyocyte degeneration in sarcoglycan-deficient Cardiomyopathy.
- Secondary Coronary artery vasospasm promotes cardiomyopathy progression.
- Sarcoglycans in vascular smooth and striated muscle.
- Cardiomyopathy is independent of skeletal muscle Disease in muscular dystrophy.
- Episodic Coronary artery vasospasm and hypertension develop in the absence of Sur2 K(ATP) channels.
- Calcium-sensitive phospholipid binding properties of normal and mutant ferlin C2 domains.
- Beta-myosin heavy chain gene mutations in familial hypertrophic Cardiomyopathy: the usual suspect?
- Genetic profiling for risk reduction in human cardiovascular Disease.
- The CO-Regulation Database (CORD): A Tool to Identify Coordinately Expressed Genes.
Schools
Albert Einstein College of Medicine of Yeshiva University
Brigham and Women39s Hospital
Harvard Med School
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac MRI (Magnetic Resonance Imaging) of Heart or Chest
- Cardiac Myocardial Perfusion Imaging
- View All
Conditions Treated
- Aneurysm and Dissection of Heart
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Dissection
- View All
Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
- Super Doctor
Education
-
Brigham and Women's Hospital -
Albert Einstein College of Medicine of Yeshiva University
Hospital
-
University of Chicago Medical Center
Drug Facts
| NPI NUMBER | 1104988658 | |
| NPPES Provider LastName | MCNALLY | |
| NPPES Provider FirstName | ELIZABETH | |
| NPPES Provider ZIPCode | 605276686 | |
| NPPES Provider State | IL | |
| Specialty Description | Cardiology | |
| Total Claim Count | 377.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1104988658 |
| Last Name Of The Provider | MCNALLY |
| First Name Of The Provider | ELIZABETH |
| View All |
Similar Doctors
|
Dr. Eric C. Svensson, MD
 This Doctor is OpenDoctor Recommended.
|
|
|
Dr. R P. Ward, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Jeanne M. Decara, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Michael Roberts, MD |
|
|
Dr. Angelo M. Scanu, MD
This Doctor is OpenDoctor Recommended.
 This doctor has more publications
|
|
|
Dr. Lissa Sugeng, MD
This Doctor is OpenDoctor Recommended.
|