Associate Professor, Dermatology, UCSF School of Medicine
Dermatology
Dr. Dennis H. Oh, MD
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Dennis H. Oh, MD is an expert in the field of Dermatology. He studied medicine at Stanford University School of Medicine. Stanford University School of Medicine is a top medical college. Stanford University School of Medicine is ranked 2 in research and 25 in primary care medicine He received awards:"Fellow (FAAD)". Dennis H. Oh is a published doctor. He published 23 publications, including: 'Transcription restores DNA repair to heterochromatin, determining regional mutation rates in cancer genomes.'
Publications
- Transcription restores DNA repair to heterochromatin, determining regional mutation rates in Cancer genomes.
- Vitamin D in cutaneous carcinogenesis: Part II.
- Vitamin D in cutaneous carcinogenesis: Part I.
- Protective role of vitamin D signaling in Skin cancer formation.
- Vitamin D Receptor Mediates DNA Repair and Is UV Inducible in Intact Epidermis but Not in Cultured Keratinocytes.
- State of teledermatology programs in the United States.
- Defective DNA repair and cell cycle arrest in cells expressing merkel cell polyomavirus T antigen.
- Targeting protein-trafficking pathways alters Melanoma treatment sensitivity.
- Functional relevance of the histone {gamma}H2Ax in the response to DNA damaging agents.
- Hereditary photodermatoses.
- The impact of store-and-forward teledermatology on Skin cancer diagnosis and treatment.
- Modulation of psoralen DNA crosslinking kinetics associated with a triplex-forming oligonucleotide.
- DNA polymerase eta reduces the gamma-H2AX response to psoralen interstrand crosslinks in human cells.
- Proficient global nucleotide excision repair in human keratinocytes but not in fibroblasts deficient in p53.
- Spatial control of reactive oxygen species formation in fibroblasts using two-photon excitation.
- American Telemedicine Association's Practice Guidelines for Teledermatology.
- The DNA damage-binding protein XPC is a frequent target for inactivation in squamous cell carcinomas.
- Differentiating human keratinocytes are deficient in p53 but retain global nucleotide excision repair following ultraviolet radiation.
- Impaired repair of cyclobutane pyrimidine dimers in human keratinocytes deficient in p53 and p63.
- gamma-H2AX formation in response to interstrand crosslinks requires XPF in human cells.
- Basal cell carcinomas arising within multiple trichoepitheliomas.
- Structural determinants of photoreactivity of triplex forming oligonucleotides conjugated to psoralens.
Schools
Stanford University
Brigham and Womens Hospital
Stanford University Mc
Procedures Preformed
- Destruction of Benign Skin Lesion
Conditions Treated
- Actinic Keratosis
- Benign Skin Lesion
- Dermatitis
- Folliculitis
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Doctors Specialties
- Dermatology
Accepted Insurances
- Aetna
- GEHA
- Covered California - Bronze PPO
- Health Net PPO
- Individual and Family PPO
- Medicare Advantage PFFS
- Mail Handlers Benefit Plan (MHBP)
- Medicare MCR
Awards
- Fellow (FAAD)
Education
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Stanford University School of Medicine
Hospital
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San Francisco VA Medical Center -
UCSF Medical Center
Drug Facts
| NPI NUMBER | 1225111511 | |
| NPPES Provider LastName | OH | |
| NPPES Provider FirstName | DENNIS | |
| NPPES Provider ZIPCode | 011990001 | |
| NPPES Provider State | MA | |
| Specialty Description | Neurosurgery | |
| Total Claim Count | 85.0 | |
| Distinct Opioid Count | 2.0 | |
| Opioid Claim Count | 34.0 | |
| Percent Opioid Claims | 40.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1225111511 |
| Last Name Of The Provider | OH |
| First Name Of The Provider | DENNIS |
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