Associate Professor, Medicine, University of Washington School of Medicine
Endocrinology
Dr. Brent E. Wisse, MD
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Brent Wisse is an expert in the field of Endocrinology. Brent Wisse is a published doctor. He published 44 publications, including: 'Induction of a glucocorticoid-sensitive F1-anti-parental mechanism that affects engraftment during graft-versus-host disease.' Doctor Brent E. Wisse, MD accepts all Medicare patients.
Publications
- Perioperative Glycemic Control During Colorectal Surgery.
- Point-of-care blood glucose measurement errors overestimate hypoglycemia rates in critically Ill patients.
- Evaluation of point-of-care blood glucose measurements in patients With diabetic ketoacidosis or hyperglycemic hyperosmolar syndrome admitted to a critical care unit.
- Hypothalamic inflammation: marker or mechanism of Obesity pathogenesis?
- In Uncontrolled Diabetes, Thyroid Hormone and Sympathetic Activators Induce Thermogenesis Without Increasing Glucose Uptake in Brown Adipose Tissue.
- BDNF Action in the Brain Attenuates Diabetic Hyperglycemia via Insulin-Independent Inhibition of Hepatic Glucose Production.
- Lipopolysaccharide-induced lung injury is independent of serum vitamin d concentration.
- Acutely decreased thermoregulatory energy expenditure or decreased activity energy expenditure both acutely reduce food intake in mice.
- Obesity is associated With hypothalamic injury in rodents and humans.
- The Impact of Inpatient Point-Of-Care Blood Glucose Quality Control Testing.
- Increased Energy Expenditure and Leptin Sensitivity Account for Low Fat Mass in Myostatin Deficient Mice.
- Identification of a physiological role for leptin in the regulation of ambulatory activity and wheel running in mice.
- Leptin activates a novel CNS mechanism for insulin-independent normalization of severe Diabetic hyperglycemia.
- Central administration of interleukin-4 exacerbates hypothalamic inflammation and weight gain during high-fat feeding.
- Leptin deficiency causes insulin resistance induced by uncontrolled Diabetes.
- Cultured hypothalamic neurons are resistant to inflammation and insulin resistance induced by saturated fatty acids.
- Mice lacking hepatic lipase are lean and protected against diet-induced Obesity and hepatic steatosis.
- Hypothalamic inflammation and energy homeostasis: resolving the paradox.
- Atypical protein kinase C activity in the hypothalamus is required for lipopolysaccharide-mediated sickness responses.
- Does hypothalamic inflammation cause Obesity?
- Forebrain melanocortin signaling enhances the hindbrain satiety response to CCK-8.
- Vascular inflammation, insulin resistance, and reduced nitric oxide production precede the onset of peripheral insulin resistance.
- Physiology. An integrative view of Obesity.
- Evidence that lipopolysaccharide-induced anorexia depends upon central, rather than peripheral, inflammatory signals.
- Current controversy related to glucocorticoid and insulin therapy in the intensive care unit.
- MyD88 is a key mediator of anorexia, but not weight loss, induced by lipopolysaccharide and interleukin-1 beta.
- Role of hypothalamic interleukin-1beta (IL-1beta) in regulation of energy homeostasis by melanocortins.
- Physiological regulation of hypothalamic IL-1beta gene expression by leptin and glucocorticoids: implications for energy homeostasis.
- The inflammatory syndrome: the role of adipose tissue cytokines in metabolic Disorders linked to obesity.
- The skinny on neurotrophins.
- Melanocortin signaling and anorexia in chronic Disease states.
- Efficacy of Diabetes nurse expert team program to improve nursing confidence and expertise in caring for hospitalized patients with Diabetes mellitus.
- Recovery from prolonged metabolic acidosis due to accidental ethylene glycol poisoning.
- Induction of a glucocorticoid-sensitive F1-anti-parental mechanism that affects engraftment during graft-versus-host Disease.
- Images in clinical medicine. Severe mitral regurgitation.
- Malformations in offspring of Diabetic rats: morphometric analysis of neural crest-derived organs and effects of maternal vitamin E treatment.
- Improving insulin ordering safely: the development of an inpatient glycemic control program.
- Identification of body fat mass as a major determinant of metabolic rate in mice.
- Central interleukin-1 (IL1) signaling is required for pharmacological, but not physiological, effects of leptin on energy balance.
- Effect of prolonged moderate and severe energy restriction and refeeding on plasma leptin concentrations in obese women.
- Role of melanocortins in control of Obesity.
- A preprandial rise in plasma ghrelin levels suggests a role in meal initiation in humans.
- Reversal of cancer anorexia by blockade of central melanocortin receptors in rats.
Conditions Treated
- Adrenal Insufficiency
- Calcium Metabolism Disorders
- Diabetes Insipidus
- Diabetes Type 1
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Doctors Specialties
- Endocrinology
Accepted Insurances
Education
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University of Washington -
McGill University Faculty of Medicine
Hospital
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UW Medicine/Harborview Medical Center -
University of Washington Medical Center
Drug Facts
| NPI NUMBER | 1154543932 | |
| NPPES Provider LastName | WISSE | |
| NPPES Provider FirstName | BRENT | |
| NPPES Provider ZIPCode | 981042420 | |
| NPPES Provider State | WA | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 104.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1154543932 |
| Last Name Of The Provider | WISSE |
| First Name Of The Provider | BRENT |
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