Holtom Garrett Professor of Neurology, Director of the Multiple Sclerosis Center and Neuroimmunology Program, University of Michigan
Neurology
Dr. Benjamin M. Segal, MD
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Benjamin Segal works in the field of Neurology. He attended Warren Alpert Medical School of Brown University. He received 1 awards: "CMS Meaningful Use Stage 1 Certification". Benjamin M. Segal is a published doctor. He has 40 papers published. The most current publication "Stable biomarker for plastic microglia." He accepts all Medicare patients.
Publications
- Stable biomarker for plastic microglia.
- Th Cell Diversity in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis.
- Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.
- The dual roles of immunity in ALS: Injury overrides protection.
- Neutrophil-related factors as biomarkers in EAE and MS.
- Dysregulation of the IL-23/IL-17 axis and myeloid factors in secondary progressive MS.
- Underrecognition of Sleep Disorders in Patients with Multiple Sclerosis.
- Hypnotic use and fatigue in Multiple sclerosis.
- Site-Specific Chemokine Expression Regulates Central Nervous System Inflammation and Determines Clinical Phenotype in Autoimmune Encephalomyelitis.
- Notch Signaling Regulates T Cell Accumulation and Function in the Central Nervous System during Experimental Autoimmune Encephalomyelitis.
- Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent.
- Highly polarized Th17 cells induce EAE via a T-bet independent mechanism.
- B-Cell Targeting Agents in the Treatment of Multiple Sclerosis.
- Virus-induced CD8+ T cells accelerate the onset of experimental autoimmune encephalomyelitis: Implications for how viral infections might trigger Multiple sclerosis ex...
- Fatigue, tiredness, lack of energy, and sleepiness in Multiple sclerosis patients referred for clinical polysomnography.
- Experimental autoimmune encephalomyelitis.
- Sleep-disordered breathing in Multiple sclerosis.
- Differences in Diffusion Tensor Imaging-Derived Metrics in the Corpus Callosum of Patients With Multiple Sclerosis Without and With Gadolinium-Enhancing Cerebral Lesions.
- The unwavering commitment of regulatory T cells in the suppression of autoimmune encephalomyelitis: Another aspect of immune privilege in the CNS.
- T(H)17 cytokines in autoimmune neuro-inflammation.
- Progressive decline in fractional anisotropy on serial DTI examinations of the corpus callosum: a putative marker of Disease activity and progression in SPMS.
- IL-23 modulated myelin-specific T cells induce EAE via an IFNγ driven, IL-17 independent pathway.
- The lymphoid chemokine, CXCL13, is dispensable for the initial recruitment of B cells to the acutely inflamed central nervous system.
- GM-CSF-dependent, CD103+ dermal dendritic cells play a critical role in Th effector cell differentiation after subcutaneous immunization.
- Circulating Ly-6C+ myeloid precursors migrate to the CNS and play a pathogenic role during autoimmune demyelinating Disease.
- Treatment of CNS sarcoidosis with infliximab and mycophenolate mofetil.
- IL-12- and IL-23-modulated T cells induce distinct types of EAE based on histology, CNS chemokine profile, and response to cytokine inhibition.
- Th17 and Th1 responses directed against the immunizing epitope, as opposed to secondary epitopes, dominate the autoimmune repertoire during relapses of experimental au...
- Cutting edge: CNS CD11c+ cells from mice with encephalomyelitis polarize Th17 cells and support CD25+CD4+ T cell-mediated immunosuppression, suggesting dual roles in t...
- CXC chemokine ligand 13 plays a role in experimental autoimmune encephalomyelitis.
- Increased rejection of primary tumors in mice lacking B cells: inhibition of anti-tumor CTL and TH1 cytokine responses by B cells.
- Sublime diagnosis of Lyme neuroborreliosis.
- Cutting edge: IL-12 induces CD4+CD25- T cell activation in the presence of T regulatory cells.
- Experimental autoimmune encephalomyelitis: cytokines, effector T cells, and antigen-presenting cells in a prototypical Th1-mediated autoimmune Disease.
- Activation of APCs through CD40 or Toll-like receptor 9 overcomes tolerance and precipitates autoimmune Disease.
- Fatigue in Multiple sclerosis: current understanding and future directions.
- Cutting Edge: IL-10-producing CD4+ T cells mediate tumor rejection.
- Obstructive Sleep Apnea is an Under-Recognized and Consequential Morbidity in Multiple Sclerosis.
- Obstructive sleep apnea and fatigue in patients with Multiple sclerosis.
Schools
Brown University Alpert Medical School
Procedures Preformed
- EEG (Electroencephalogram)
Conditions Treated
- Ataxia
- Cerebral Artery Thrombosis
- Cerebrovascular Disease
- Convulsions
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Doctors Specialties
- Neurology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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New York Presbyterian Hospital - Cornell Campus -
University of Chicago -
Warren Alpert Medical School of Brown University
Hospital
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University of Michigan Hospitals and Health Centers
Drug Facts
| NPI NUMBER | 1740299361 | |
| NPPES Provider LastName | SEGAL | |
| NPPES Provider FirstName | BENJAMIN | |
| NPPES Provider ZIPCode | 481095322 | |
| NPPES Provider State | MI | |
| Specialty Description | Neurology | |
| Total Claim Count | 264.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 25.0 | |
| Percent Opioid Claims | 9.47 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1740299361 |
| Last Name Of The Provider | SEGAL |
| First Name Of The Provider | BENJAMIN |
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